David S. Wilkes
Affiliations: | Indiana University, Bloomington, Bloomington, IN, United States |
Area:
Immunology, Microbiology BiologyGoogle:
"David Wilkes"Children
Sign in to add traineeKena A. Swanson | grad student | 2003 | Indiana University Bloomington |
Allison T. Thiele | grad student | 2005 | Indiana University Bloomington |
Tina L. Sumpter | grad student | 2006 | Indiana University Bloomington |
Heather L. Benson | grad student | 2009 | Indiana University Bloomington |
Jeremy M. Lott | grad student | 2012 | Indiana University Bloomington |
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Publications
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Hata A, Guo Y, Miller AE, et al. (2022) Loss of stromal cell Thy-1 plays a critical role in lipopolysaccharide induced chronic lung allograft dysfunction. The Journal of Heart and Lung Transplantation : the Official Publication of the International Society For Heart Transplantation |
Zaffiri L, Shah RJ, Stearman RS, et al. (2019) Collagen type-V is a danger signal associated with primary graft dysfunction in lung transplantation. Transplant Immunology. 101224 |
Guo Y, Wang Q, Li D, et al. (2019) Vendor-Specific Microbiome Controls both Acute and Chronic Murine Lung Allograft Rejection by Altering CD4 Foxp3 Regulatory T Cell Levels. American Journal of Transplantation : Official Journal of the American Society of Transplantation and the American Society of Transplant Surgeons |
Wilkes DS, Heidler KM, Yasufuku K, et al. (2019) Cell-mediated immunity to collagen V in lung transplant recipients: correlation with collagen V release into BAL fluid. The Journal of Heart and Lung Transplantation : the Official Publication of the International Society For Heart Transplantation. 20: 167 |
Vittal R, Mickler EA, Fisher AJ, et al. (2018) Correction: Type V Collagen Induced Tolerance Suppresses Collagen Deposition, TGF-β and Associated Transcripts in Pulmonary Fibrosis. Plos One. 13: e0209107 |
Cantu E, Diamond JM, Suzuki Y, et al. (2017) Quantitative Evidence for Revising the Definition of Primary Graft Dysfunction After Lung Transplant. American Journal of Respiratory and Critical Care Medicine |
Porteous MK, Lee JC, Lederer DJ, et al. (2017) Clinical Risk Factors and Prognostic Model for Primary Graft Dysfunction after Lung Transplantation in Patients with Pulmonary Hypertension. Annals of the American Thoracic Society |
Forcade E, Paz K, Flynn R, et al. (2017) An activated Th17-prone T cell subset involved in chronic graft-versus-host disease sensitive to pharmacological inhibition. Jci Insight. 2 |
Lama VN, Belperio JA, Christie JD, et al. (2017) Models of Lung Transplant Research: a consensus statement from the National Heart, Lung, and Blood Institute workshop. Jci Insight. 2 |
Sullivan JA, Jankowska-Gan E, Hegde S, et al. (2016) Th17 responses to Col V, kα-1-tubulin and vimentin are present early in human development and persist lifelong. American Journal of Transplantation : Official Journal of the American Society of Transplantation and the American Society of Transplant Surgeons |