Year |
Citation |
Score |
2023 |
Groome JR. Historical Perspective of the Characterization of Conotoxins Targeting Voltage-Gated Sodium Channels. Marine Drugs. 21. PMID 37103349 DOI: 10.3390/md21040209 |
0.347 |
|
2021 |
Zhu Q, Du Y, Nomura Y, Gao R, Cang Z, Wei GW, Gordon D, Gurevitz M, Groome J, Dong K. Charge substitutions at the voltage-sensing module of domain III enhance actions of site-3 and site-4 toxins on an insect sodium channel. Insect Biochemistry and Molecular Biology. 103625. PMID 34358664 DOI: 10.1016/j.ibmb.2021.103625 |
0.427 |
|
2020 |
Groome JR, Bayless-Edwards L. Roles for Countercharge in the Voltage Sensor Domain of Ion Channels. Frontiers in Pharmacology. 11: 160. PMID 32180723 DOI: 10.3389/fphar.2020.00160 |
0.37 |
|
2019 |
Groome JR, Wheeler A, Camp R. Domain I Countercharges Limit Slow Inactivation in hNav1.4 Sodium Channels Biophysical Journal. 116: 390a. DOI: 10.1016/J.BPJ.2018.11.2113 |
0.342 |
|
2018 |
Bayless-Edwards L, Winston V, Lehmann-Horn F, Arinze P, Groome JR, Jurkat-Rott K. Na1.4 DI-S4 periodic paralysis mutation R222W enhances inactivation and promotes leak current to attenuate action potentials and depolarize muscle fibers. Scientific Reports. 8: 10372. PMID 29991727 DOI: 10.1038/S41598-018-28594-5 |
0.416 |
|
2018 |
Groome JR, Moreau A, Delemotte L. Gating Pore Currents in Sodium Channels. Handbook of Experimental Pharmacology. 246: 371-399. PMID 28965172 DOI: 10.1007/164_2017_54 |
0.474 |
|
2017 |
Groome JR, Camp R. Role for Fast Inactivation in Domain I of Voltage Gated Sodium Channels Biophysical Journal. 112: 103a. DOI: 10.1016/j.bpj.2016.11.589 |
0.404 |
|
2016 |
Bednarz M, Stunnenberg BC, Kusters B, Kamsteeg EJ, Saris CG, Groome J, Winston V, Meola G, Jurkat-Rott K, Voermans NC. A novel Ile1455Thr variant in the skeletal muscle sodium channel alpha-subunit in a patient with a severe adult-onset proximal myopathy with electrical myotonia and a patient with mild paramyotonia phenotype. Neuromuscular Disorders : Nmd. PMID 28024841 DOI: 10.1016/J.Nmd.2016.09.023 |
0.51 |
|
2015 |
Groome JR, Winston V. Correction: S1-S3 counter charges in the voltage sensor module of a mammalian sodium channel regulate fast inactivation. The Journal of General Physiology. PMID 26573623 DOI: 10.1085/Jgp.20121093510232015C |
0.522 |
|
2015 |
Groome JR. Voltage Sensor Domains and Closed-State Inactivation in Sodium Channels Biophysical Journal. 108: 572a. DOI: 10.1016/j.bpj.2014.11.3130 |
0.353 |
|
2014 |
Groome JR, Jurkat-Rott K, Lehmann-Horn F. Domain III S4 in closed-state fast inactivation: insights from a periodic paralysis mutation. Channels (Austin, Tex.). 8: 467-71. PMID 25483590 DOI: 10.4161/19336950.2014.958924 |
0.577 |
|
2014 |
Groome JR. The voltage sensor module in sodium channels. Handbook of Experimental Pharmacology. 221: 7-31. PMID 24737230 DOI: 10.1007/978-3-642-41588-3_2 |
0.383 |
|
2014 |
Groome JR, Lehmann-Horn F, Fan C, Wolf M, Winston V, Merlini L, Jurkat-Rott K. NaV1.4 mutations cause hypokalaemic periodic paralysis by disrupting IIIS4 movement during recovery. Brain : a Journal of Neurology. 137: 998-1008. PMID 24549961 DOI: 10.1093/Brain/Awu015 |
0.517 |
|
2013 |
Fan C, Lehmann-Horn F, Weber MA, Bednarz M, Groome JR, Jonsson MK, Jurkat-Rott K. Transient compartment-like syndrome and normokalaemic periodic paralysis due to a Ca(v)1.1 mutation. Brain : a Journal of Neurology. 136: 3775-86. PMID 24240197 DOI: 10.1093/brain/awt300 |
0.458 |
|
2013 |
Groome JR, Winston V. S1-S3 counter charges in the voltage sensor module of a mammalian sodium channel regulate fast inactivation. The Journal of General Physiology. 141: 601-18. PMID 23589580 DOI: 10.1085/Jgp.201210935 |
0.506 |
|
2012 |
Jurkat-Rott K, Groome J, Lehmann-Horn F. Pathophysiological role of omega pore current in channelopathies. Frontiers in Pharmacology. 3: 112. PMID 22701429 DOI: 10.3389/fphar.2012.00112 |
0.464 |
|
2012 |
Groome JR, Winston V. Voltage Sensor Module in NaV1.4: S1-S3 Charges Regulate Fast Inactivation Biophysical Journal. 102: 326a. DOI: 10.1016/J.Bpj.2011.11.1787 |
0.488 |
|
2011 |
Groome J, Lehmann-Horn F, Holzherr B. Open- and closed-state fast inactivation in sodium channels: differential effects of a site-3 anemone toxin. Channels (Austin, Tex.). 5: 65-78. PMID 21099342 DOI: 10.4161/chan.5.1.14031 |
0.552 |
|
2011 |
Groome JR, Wagley P. DIII - DIV Linker Charge Mutations Differentially Affect Open and Closed State Fast Inactivation in Nav1.4 Biophysical Journal. 100: 423a. DOI: 10.1016/j.bpj.2010.12.2502 |
0.494 |
|
2010 |
Du Y, Song W, Groome JR, Nomura Y, Luo N, Dong K. A negative charge in transmembrane segment 1 of domain II of the cockroach sodium channel is critical for channel gating and action of pyrethroid insecticides. Toxicology and Applied Pharmacology. 247: 53-9. PMID 20561903 DOI: 10.1016/J.Taap.2010.05.016 |
0.489 |
|
2009 |
Groome JR. Charge Immobilization From The Open And Closed States Of Voltage-Gated Sodium Channels Biophysical Journal. 96: 248a. DOI: 10.1016/J.BPJ.2008.12.1223 |
0.504 |
|
2008 |
Groome JR, Larsen MF, Coonts A. Differential effects of paramyotonia congenita mutations F1473S and F1705I on sodium channel gating. Channels (Austin, Tex.). 2: 39-50. PMID 18690054 DOI: 10.4161/chan.2.1.6051 |
0.521 |
|
2007 |
Groome JR, Dice MC, Fujimoto E, Ruben PC. Charge immobilization of skeletal muscle Na+ channels: role of residues in the inactivation linker. Biophysical Journal. 93: 1519-33. PMID 17513361 DOI: 10.1529/biophysj.106.102079 |
0.68 |
|
2007 |
Groome JR, Alexander HM, Fujimoto E, Sherry M, Petty D. Central charged residues in DIIIS4 regulate deactivation gating in skeletal muscle sodium channels. Cellular and Molecular Neurobiology. 27: 87-106. PMID 17151947 DOI: 10.1007/s10571-006-9120-2 |
0.437 |
|
2005 |
Groome JR, Fujimoto E, Ruben PC. K-aggravated myotonia mutations at residue G1306 differentially alter deactivation gating of human skeletal muscle sodium channels. Cellular and Molecular Neurobiology. 25: 1075-92. PMID 16392038 DOI: 10.1007/s10571-005-8057-1 |
0.653 |
|
2004 |
Dice MS, Abbruzzese JL, Wheeler JT, Groome JR, Fujimoto E, Ruben PC. Temperature-sensitive defects in paramyotonia congenita mutants R1448C and T1313M. Muscle & Nerve. 30: 277-88. PMID 15318338 DOI: 10.1002/Mus.20080 |
0.594 |
|
2003 |
Groome JR, Fujimoto E, Ruben PC. Negative charges in the DIII-DIV linker of human skeletal muscle Na+ channels regulate deactivation gating. The Journal of Physiology. 548: 85-96. PMID 12588896 DOI: 10.1113/jphysiol.2002.033084 |
0.671 |
|
2002 |
Groome J, Fujimoto E, Walter L, Ruben P. Outer and central charged residues in DIVS4 of skeletal muscle sodium channels have differing roles in deactivation. Biophysical Journal. 82: 1293-307. PMID 11867446 DOI: 10.1016/S0006-3495(02)75485-4 |
0.61 |
|
2000 |
Groome JR, Fujimoto E, Ruben PC. The delay in recovery from fast inactivation in skeletal muscle sodium channels is deactivation. Cellular and Molecular Neurobiology. 20: 521-7. PMID 10901271 DOI: 10.1023/A:1007040731407 |
0.665 |
|
1999 |
Groome JR, Fujimoto E, George AL, Ruben PC. Differential effects of homologous S4 mutations in human skeletal muscle sodium channels on deactivation gating from open and inactivated states. The Journal of Physiology. 516: 687-98. PMID 10200418 DOI: 10.1111/j.1469-7793.1999.0687u.x |
0.674 |
|
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