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Anny Shai, Ph.D.

Affiliations: 
2008 University of Wisconsin, Madison, Madison, WI 
Area:
Oncology, Molecular Biology
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"Anny Shai"
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Paul F. Lambert grad student 2008 UW Madison
 (The trimeric complex in HPV: Molecular mechanisms of HPV16 E6, E6AP and p53 in HPV -associated cancers.)

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Publications

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Shai A, Dankort D, Juan J, et al. (2015) TP53 Silencing Bypasses Growth Arrest of BRAFV600E-Induced Lung Tumor Cells In a Two-Switch Model of Lung Tumorigenesis. Cancer Research
Shai A, Pitot HC, Lambert PF. (2010) E6-associated protein is required for human papillomavirus type 16 E6 to cause cervical cancer in mice. Cancer Research. 70: 5064-73
Maufort JP, Shai A, Pitot HC, et al. (2010) A role for HPV16 E5 in cervical carcinogenesis. Cancer Research. 70: 2924-31
Zehbe I, Richard C, DeCarlo CA, et al. (2009) Human papillomavirus 16 E6 variants differ in their dysregulation of human keratinocyte differentiation and apoptosis. Virology. 383: 69-77
Chung SH, Wiedmeyer K, Shai A, et al. (2008) Requirement for estrogen receptor alpha in a mouse model for human papillomavirus-associated cervical cancer. Cancer Research. 68: 9928-34
Shai A, Pitot HC, Lambert PF. (2008) p53 Loss synergizes with estrogen and papillomaviral oncogenes to induce cervical and breast cancers. Cancer Research. 68: 2622-31
Massimi P, Shai A, Lambert P, et al. (2008) HPV E6 degradation of p53 and PDZ containing substrates in an E6AP null background Oncogene. 27: 1800-1804
Shai A, Brake T, Somoza C, et al. (2007) The human papillomavirus E6 oncogene dysregulates the cell cycle and contributes to cervical carcinogenesis through two independent activities. Cancer Research. 67: 1626-35
Shai A, Nguyen ML, Wagstaff J, et al. (2007) HPV16 E6 confers p53-dependent and p53-independent phenotypes in the epidermis of mice deficient for E6AP. Oncogene. 26: 3321-8
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