| Year |
Citation |
Score |
| 2017 |
Benischke AS, Vasanth S, Miyai T, Katikireddy KR, White T, Chen Y, Halilovic A, Price M, Price F, Liton PB, Jurkunas UV. Activation of mitophagy leads to decline in Mfn2 and loss of mitochondrial mass in Fuchs endothelial corneal dystrophy. Scientific Reports. 7: 6656. PMID 28751712 DOI: 10.1038/S41598-017-06523-2 |
0.315 |
|
| 2016 |
Halilovic A, Schmedt T, Benischke AS, Hamill CE, Chen Y, Santos JH, Jurkunas UV. Menadione-induced DNA damage leads to mitochondrial dysfunction and fragmentation during rosette formation in Fuchs Endothelial Corneal Dystrophy. Antioxidants & Redox Signaling. PMID 26935406 DOI: 10.1089/Ars.2015.6532 |
0.346 |
|
| 2011 |
Marrazzo G, Bellner L, Halilovic A, Li Volti G, Drago F, Dunn MW, Schwartzman ML. The role of neutrophils in corneal wound healing in HO-2 null mice. Plos One. 6: e21180. PMID 21695050 DOI: 10.1371/Journal.Pone.0021180 |
0.606 |
|
| 2011 |
Bellner L, Patil KA, Castellano K, Halilovic A, Dunn MW, Schwartzman ML. Targeted suppression of HO-2 gene expression impairs the innate anti-inflammatory and repair responses of the cornea to injury. Molecular Vision. 17: 1144-52. PMID 21552471 |
0.605 |
|
| 2011 |
Halilovic A, Patil KA, Bellner L, Marrazzo G, Castellano K, Cullaro G, Dunn MW, Schwartzman ML. Knockdown of heme oxygenase-2 impairs corneal epithelial cell wound healing. Journal of Cellular Physiology. 226: 1732-40. PMID 21506105 DOI: 10.1002/Jcp.22502 |
0.589 |
|
| 2009 |
Bellner L, Martinelli L, Halilovic A, Patil K, Puri N, Dunn MW, Regan RF, Schwartzman ML. Heme oxygenase-2 deletion causes endothelial cell activation marked by oxidative stress, inflammation, and angiogenesis. The Journal of Pharmacology and Experimental Therapeutics. 331: 925-32. PMID 19773531 DOI: 10.1124/Jpet.109.158352 |
0.561 |
|
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