Year |
Citation |
Score |
2015 |
Burris SK, Wang Q, Bulley S, Neeb ZP, Jaggar JH. 9-Phenanthrol inhibits recombinant and arterial myocyte TMEM16A channels. British Journal of Pharmacology. 172: 2459-68. PMID 25573456 DOI: 10.1111/Bph.13077 |
0.428 |
|
2012 |
Adebiyi A, Thomas-Gatewood CM, Leo MD, Kidd MW, Neeb ZP, Jaggar JH. An elevation in physical coupling of type 1 inositol 1,4,5-trisphosphate (IP3) receptors to transient receptor potential 3 (TRPC3) channels constricts mesenteric arteries in genetic hypertension. Hypertension. 60: 1213-9. PMID 23045459 DOI: 10.1161/Hypertensionaha.112.198820 |
0.454 |
|
2012 |
Bulley S, Neeb ZP, Burris SK, Bannister JP, Thomas-Gatewood CM, Jangsangthong W, Jaggar JH. TMEM16A/ANO1 channels contribute to the myogenic response in cerebral arteries. Circulation Research. 111: 1027-36. PMID 22872152 DOI: 10.1161/Circresaha.112.277145 |
0.437 |
|
2012 |
Trask AJ, Katz PS, Kelly AP, Galantowicz ML, Cismowski MJ, West TA, Neeb ZP, Berwick ZC, Goodwill AG, Alloosh M, Tune JD, Sturek M, Lucchesi PA. Dynamic micro- and macrovascular remodeling in coronary circulation of obese Ossabaw pigs with metabolic syndrome. Journal of Applied Physiology (Bethesda, Md. : 1985). 113: 1128-40. PMID 22837170 DOI: 10.1152/Japplphysiol.00604.2012 |
0.62 |
|
2011 |
Thomas-Gatewood C, Neeb ZP, Bulley S, Adebiyi A, Bannister JP, Leo MD, Jaggar JH. TMEM16A channels generate Ca²⁺-activated Cl⁻ currents in cerebral artery smooth muscle cells. American Journal of Physiology. Heart and Circulatory Physiology. 301: H1819-27. PMID 21856902 DOI: 10.1152/Ajpheart.00404.2011 |
0.515 |
|
2011 |
Kreutz RP, Alloosh M, Mansour K, Neeb Z, Kreutz Y, Flockhart DA, Sturek M. Morbid obesity and metabolic syndrome in Ossabaw miniature swine are associated with increased platelet reactivity. Diabetes, Metabolic Syndrome and Obesity: Targets and Therapy. 4: 99-105. PMID 21660293 DOI: 10.2147/Dmso.S17105 |
0.594 |
|
2011 |
Bannister JP, Thomas-Gatewood CM, Neeb ZP, Adebiyi A, Cheng X, Jaggar JH. Ca(V)1.2 channel N-terminal splice variants modulate functional surface expression in resistance size artery smooth muscle cells. The Journal of Biological Chemistry. 286: 15058-66. PMID 21357696 DOI: 10.1074/Jbc.M110.182816 |
0.416 |
|
2010 |
Long X, Mokelke EA, Neeb ZP, Alloosh M, Edwards JM, Sturek M. Adenosine receptor regulation of coronary blood flow in Ossabaw miniature swine. The Journal of Pharmacology and Experimental Therapeutics. 335: 781-7. PMID 20855445 DOI: 10.1124/Jpet.110.170803 |
0.633 |
|
2010 |
Neeb ZP, Edwards JM, Alloosh M, Long X, Mokelke EA, Sturek M. Metabolic syndrome and coronary artery disease in Ossabaw compared with Yucatan swine. Comparative Medicine. 60: 300-15. PMID 20819380 |
0.699 |
|
2010 |
Zhao G, Neeb ZP, Dennis Leo M, Pachuau J, Adebiyi A, Ouyang K, Chen J, Jaggar JH. Type 1 IP3 receptors activate BKC3 channels via local molecular coupling in arterial smooth muscle cells Journal of General Physiology. 136: 283-291. PMID 20713546 DOI: 10.1085/Jgp.201010453 |
0.448 |
|
2010 |
Payne GA, Borbouse L, Kumar S, Neeb Z, Alloosh M, Sturek M, Tune JD. Epicardial perivascular adipose-derived leptin exacerbates coronary endothelial dysfunction in metabolic syndrome via a protein kinase C-β pathway Arteriosclerosis, Thrombosis, and Vascular Biology. 30: 1711-1717. PMID 20576943 DOI: 10.1161/Atvbaha.110.210070 |
0.652 |
|
2010 |
Borbouse L, Dick GM, Payne GA, Berwick ZC, Neeb ZP, Alloosh M, Bratz IN, Sturek M, Tune JD. Metabolic syndrome reduces the contribution of K+ channels to ischemic coronary vasodilation. American Journal of Physiology. Heart and Circulatory Physiology. 298: H1182-9. PMID 20118408 DOI: 10.1152/Ajpheart.00888.2009 |
0.645 |
|
2010 |
Borbouse L, Dick GM, Payne GA, Payne BD, Svendsen MC, Neeb ZP, Alloosh M, Bratz IN, Sturek M, Tune JD. Contribution of BK(Ca) channels to local metabolic coronary vasodilation: Effects of metabolic syndrome. American Journal of Physiology. Heart and Circulatory Physiology. 298: H966-73. PMID 20044440 DOI: 10.1152/Ajpheart.00876.2009 |
0.672 |
|
2010 |
Edwards JM, Neeb ZP, Alloosh MA, Long X, Bratz IN, Peller CR, Byrd JP, Kumar S, Obukhov AG, Sturek M. Exercise training decreases store-operated Ca2+entry associated with metabolic syndrome and coronary atherosclerosis. Cardiovascular Research. 85: 631-40. PMID 19744946 DOI: 10.1093/Cvr/Cvp308 |
0.683 |
|
2009 |
Borbouse L, Dick GM, Asano S, Bender SB, Dincer UD, Payne GA, Neeb ZP, Bratz IN, Sturek M, Tune JD. Impaired function of coronary BK(Ca) channels in metabolic syndrome. American Journal of Physiology. Heart and Circulatory Physiology. 297: H1629-37. PMID 19749164 DOI: 10.1152/Ajpheart.00466.2009 |
0.693 |
|
2008 |
Bratz IN, Dick GM, Tune JD, Edwards JM, Neeb ZP, Dincer UD, Sturek M. Impaired capsaicin-induced relaxation of coronary arteries in a porcine model of the metabolic syndrome. American Journal of Physiology. Heart and Circulatory Physiology. 294: H2489-96. PMID 18390821 DOI: 10.1152/Ajpheart.01191.2007 |
0.682 |
|
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