Gary A. Splitter
Affiliations: | University of Wisconsin, Madison, Madison, WI |
Area:
Immunology, Microbiology BiologyGoogle:
"Gary Splitter"Children
Sign in to add traineeDohun Pyeon | grad student | 1999 | |
Xiaodi Ren | grad student | 2000 | UW Madison |
Jinkyung Ko | grad student | 2001 | UW Madison |
Aurea M. Canavessi | grad student | 2003 | UW Madison |
Angela J. Mathison | grad student | 2007 | UW Madison |
Marina A. Durward | grad student | 2011 | UW Madison |
Diogo Magnani | grad student | 2011 | UW Madison |
Erik Petersen | grad student | 2011 | UW Madison |
Christopher R. Gourley | grad student | 2012 | UW Madison |
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Publications
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Daggett J, Rogers A, Harms J, et al. (2020) Hepatic and splenic immune response during acute vs. chronic Brucella melitensis infection using in situ microscopy. Comparative Immunology, Microbiology and Infectious Diseases. 73: 101490 |
Guimarães ES, Gomes MTR, Campos PC, et al. (2019) Cyclic Dinucleotides Trigger STING-Dependent Unfolded Protein Response That Favors Bacterial Replication. Journal of Immunology (Baltimore, Md. : 1950) |
Harms JS, Khan M, Hall C, et al. (2018) Peptide Cross-Reactive MHC I Presentation Activates SIINFEKL-Specific TCR Expressing T Cells. Infection and Immunity |
Jakka P, Bhargavi B, Namani S, et al. (2018) Cytoplasmic Linker Protein CLIP170 Negatively Regulates TLR4 Signaling by Targeting the TLR Adaptor Protein TIRAP. Journal of Immunology (Baltimore, Md. : 1950). 200: 704-714 |
Costa Franco MM, Marim F, Guimarães ES, et al. (2017) Brucella abortus Triggers a cGAS-Independent STING Pathway To Induce Host Protection That Involves Guanylate-Binding Proteins and Inflammasome Activation. Journal of Immunology (Baltimore, Md. : 1950) |
Alves-Silva J, Tavares IP, Guimarães ES, et al. (2017) Modulation of Microtubule Dynamics Affects Brucella abortus Intracellular Survival, Pathogen-Containing Vacuole Maturation, and Pro-inflammatory Cytokine Production in Infected Macrophages. Frontiers in Microbiology. 8: 2217 |
Khan M, Harms JS, Marim FM, et al. (2016) The bacterial second messenger cyclic-di-GMP regulates Brucella pathogenesis and leads to altered host immune response. Infection and Immunity |
Gomes MT, Campos PC, Pereira GS, et al. (2015) TLR9 is required for MAPK/NF-κB activation but does not cooperate with TLR2 or TLR6 to induce host resistance to Brucella abortus. Journal of Leukocyte Biology |
Durward-Diioia M, Harms J, Khan M, et al. (2015) CD8+ T cell exhaustion, suppressed IFN-γ production, and delayed memory response induced by chronic Brucella melitensis infection. Infection and Immunity |
Gourley CR, Petersen E, Harms J, et al. (2015) Decreased in vivo virulence and altered gene expression by a Brucella melitensis light-sensing histidine kinase mutant. Pathogens and Disease. 73: 1-8 |