Shuangping Shi, Ph.D.
Affiliations: | 2005 | Weill Cornell Medical College, New York, NY, United States |
Area:
Immunology, Microbiology BiologyGoogle:
"Shuangping Shi"Parents
Sign in to add mentor| Carl F. Nathan | grad student | 2005 | Weill Cornell Medical College | |
| (Interplay between Mycobacterium tuberculosis and the macrophage.) | ||||
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Publications
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| Venugopal A, Bryk R, Shi S, et al. (2011) Virulence of Mycobacterium tuberculosis depends on lipoamide dehydrogenase, a member of three multienzyme complexes. Cell Host & Microbe. 9: 21-31 |
| Shi S, Ehrt S. (2006) Dihydrolipoamide acyltransferase is critical for Mycobacterium tuberculosis pathogenesis. Infection and Immunity. 74: 56-63 |
| Ding A, Yu H, Yang J, et al. (2005) Induction of macrophage-derived SLPI by Mycobacterium tuberculosis depends on TLR2 but not MyD88. Immunology. 116: 381-9 |
| Shi S, Blumenthal A, Hickey CM, et al. (2005) Expression of many immunologically important genes in Mycobacterium tuberculosis-infected macrophages is independent of both TLR2 and TLR4 but dependent on IFN-alphabeta receptor and STAT1. Journal of Immunology (Baltimore, Md. : 1950). 175: 3318-28 |
| Tian J, Bryk R, Shi S, et al. (2005) Mycobacterium tuberculosis appears to lack alpha-ketoglutarate dehydrogenase and encodes pyruvate dehydrogenase in widely separated genes. Molecular Microbiology. 57: 859-68 |
| Shi S, Nathan C, Schnappinger D, et al. (2003) MyD88 primes macrophages for full-scale activation by interferon-gamma yet mediates few responses to Mycobacterium tuberculosis. The Journal of Experimental Medicine. 198: 987-97 |
| Ehrt S, Schnappinger D, Bekiranov S, et al. (2001) Reprogramming of the macrophage transcriptome in response to interferon-gamma and Mycobacterium tuberculosis: signaling roles of nitric oxide synthase-2 and phagocyte oxidase. The Journal of Experimental Medicine. 194: 1123-40 |