Wendy L. See, Ph.D.
Affiliations: | 2009 | Weill Cornell Medical College, New York, NY, United States |
Area:
Cell BiologyGoogle:
"Wendy See"Parents
Sign in to add mentor| Marilyn Resh | grad student | 2009 | Weill Cornell Medical College | |
| (The role of p27 in gliomagenesis.) | ||||
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| Mukherjee J, Ohba S, See WL, et al. (2016) PKM2 uses control of HuR localization to regulate p27 and cell cycle progression in human glioblastoma cells. International Journal of Cancer. Journal International Du Cancer |
| Ohba S, Mukherjee J, See WL, et al. (2014) Mutant IDH1-driven cellular transformation increases RAD51-mediated homologous recombination and temozolomide resistance. Cancer Research. 74: 4836-44 |
| See WL, Tan IL, Mukherjee J, et al. (2012) Sensitivity of glioblastomas to clinically available MEK inhibitors is defined by neurofibromin 1 deficiency. Cancer Research. 72: 3350-9 |
| See WL, Heinberg AR, Holland EC, et al. (2010) p27 deficiency is associated with migration defects in PDGF-expressing gliomas in vivo. Cell Cycle (Georgetown, Tex.). 9: 1562-7 |
| See WL, Miller JP, Squatrito M, et al. (2010) Defective DNA double-strand break repair underlies enhanced tumorigenesis and chromosomal instability in p27-deficient mice with growth factor-induced oligodendrogliomas. Oncogene. 29: 1720-31 |
| Donovan S, See W, Bonifas J, et al. (2002) Hyperactivation of protein kinase B and ERK have discrete effects on survival, proliferation, and cytokine expression in Nf1-deficient myeloid cells. Cancer Cell. 2: 507-14 |