Olivia N. Chuang-Smith, Ph.D.

2009 Microbiology, Immunology and Cancer Biology University of Minnesota, Twin Cities, Minneapolis, MN 
Microbiology Biology
"Olivia Chuang-Smith"


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Gary M. Dunny grad student 2009 UMN
 (Enterococcus faecalis aggregation substance (Asc10) as liaison between bacterium and heart valve in endocarditis.)
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Colomer-Winter C, Gaca AO, Chuang-Smith ON, et al. (2018) Basal levels of (p)ppGpp differentially affect the pathogenesis of infective endocarditis in Enterococcus faecalis. Microbiology (Reading, England)
Schlievert PM, Merriman JA, Salgado-Pabón W, et al. (2013) Menaquinone analogs inhibit growth of bacterial pathogens. Antimicrobial Agents and Chemotherapy. 57: 5432-7
Frank KL, Guiton PS, Barnes AM, et al. (2013) AhrC and Eep are biofilm infection-associated virulence factors in Enterococcus faecalis. Infection and Immunity. 81: 1696-708
Mattis DM, Spaulding AR, Chuang-Smith ON, et al. (2013) Engineering a soluble high-affinity receptor domain that neutralizes staphylococcal enterotoxin C in rabbit models of disease Protein Engineering, Design and Selection. 26: 133-142
Spaulding AR, Satterwhite EA, Lin YC, et al. (2012) Comparison of Staphylococcus aureus strains for ability to cause infective endocarditis and lethal sepsis in rabbits. Frontiers in Cellular and Infection Microbiology. 2: 18
Fan J, Zhang Y, Chuang-Smith ON, et al. (2012) Ecto-5'-nucleotidase: a candidate virulence factor in Streptococcus sanguinis experimental endocarditis. Plos One. 7: e38059
Wilson GJ, Seo KS, Cartwright RA, et al. (2011) A novel core genome-encoded superantigen contributes to lethality of community-associated MRSA necrotizing pneumonia. Plos Pathogens. 7: e1002271
Chuang-Smith ON, Wells CL, Henry-Stanley MJ, et al. (2010) Acceleration of Enterococcus faecalis biofilm formation by aggregation substance expression in an ex vivo model of cardiac valve colonization. Plos One. 5: e15798
Schlievert PM, Chuang-Smith ON, Peterson ML, et al. (2010) Enterococcus faecalis endocarditis severity in rabbits is reduced by IgG Fabs interfering with aggregation substance. Plos One. 5
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