Matthew D. Westfall, Ph.D.

Affiliations: 
2003 Vanderbilt University, Nashville, TN 
Area:
Biochemistry, Cell Biology
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"Matthew Westfall"

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Jennifer A. Pietenpol grad student 2003 Vanderbilt
 (Analysis of p63 in keratinocyte growth and differentiation: Identification of DeltaNp63alpha as a transcriptional repressor.)
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Publications

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Westfall MD, Joyner AS, Barbieri CE, et al. (2005) Ultraviolet radiation induces phosphorylation and ubiquitin-mediated degradation of DeltaNp63alpha. Cell Cycle (Georgetown, Tex.). 4: 710-6
Maas K, Westfall M, Pietenpol J, et al. (2005) Reduced p53 in peripheral blood mononuclear cells from patients with rheumatoid arthritis is associated with loss of radiation-induced apoptosis. Arthritis and Rheumatism. 52: 1047-57
Sniezek JC, Matheny KE, Westfall MD, et al. (2004) Dominant negative p63 isoform expression in head and neck squamous cell carcinoma. The Laryngoscope. 114: 2063-72
Stein S, Thomas EK, Herzog B, et al. (2004) NDRG1 is necessary for p53-dependent apoptosis. The Journal of Biological Chemistry. 279: 48930-40
Westfall MD, Pietenpol JA. (2004) p63: Molecular complexity in development and cancer. Carcinogenesis. 25: 857-64
Massion PP, Taflan PM, Jamshedur Rahman SM, et al. (2003) Significance of p63 amplification and overexpression in lung cancer development and prognosis. Cancer Research. 63: 7113-21
Westfall MD, Mays DJ, Sniezek JC, et al. (2003) The Delta Np63 alpha phosphoprotein binds the p21 and 14-3-3 sigma promoters in vivo and has transcriptional repressor activity that is reduced by Hay-Wells syndrome-derived mutations. Molecular and Cellular Biology. 23: 2264-76
Stewart ZA, Westfall MD, Pietenpol JA. (2003) Cell-cycle dysregulation and anticancer therapy. Trends in Pharmacological Sciences. 24: 139-45
Eichten A, Westfall M, Pietenpol JA, et al. (2002) Stabilization and functional impairment of the tumor suppressor p53 by the human papillomavirus type 16 E7 oncoprotein. Virology. 295: 74-85
Flatt PM, Polyak K, Tang LJ, et al. (2000) p53-dependent expression of PIG3 during proliferation, genotoxic stress, and reversible growth arrest. Cancer Letters. 156: 63-72
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