Shilpa P. Deshpande, Ph.D.

2001 The University of Tennessee, Knoxville, TN, United States 
Microbiology Biology, Immunology
"Shilpa Deshpande"


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Barry T. Rouse grad student 2001 University of Tennessee
 (Pathogenic mechanisms involved in the ocular immunoinflammatory lesions: Herpetic stromal keratitis.)
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Deshpande S, Banerjee K, Biswas PS, et al. (2004) Herpetic eye disease: immunopathogenesis and therapeutic measures. Expert Reviews in Molecular Medicine. 6: 1-14
Deshpande SP, Zheng M, Lee S, et al. (2002) Mechanisms of pathogenesis in herpetic immunoinflammatory ocular lesions. Veterinary Microbiology. 86: 17-26
Deshpande S, Zheng M, Lee S, et al. (2001) Bystander activation involving T lymphocytes in herpetic stromal keratitis. Journal of Immunology (Baltimore, Md. : 1950). 167: 2902-10
Deshpande SP, Lee S, Zheng M, et al. (2001) Herpes simplex virus-induced keratitis: evaluation of the role of molecular mimicry in lesion pathogenesis. Journal of Virology. 75: 3077-88
Deshpande SP, Kumaraguru U, Rouse BT. (2000) Why do we lack an effective vaccine against herpes simplex virus infections? Microbes and Infection / Institut Pasteur. 2: 973-8
Deshpande SP, Kumaraguru U, Rouse BT. (2000) Dual role of B cells in mediating innate and acquired immunity to herpes simplex virus infections. Cellular Immunology. 202: 79-87
Deshpande SP, Zheng M, Daheshia M, et al. (2000) Pathogenesis of herpes simplex virus-induced ocular immunoinflammatory lesions in B-cell-deficient mice. Journal of Virology. 74: 3517-24
Gangappa S, Deshpande SP, Rouse BT. (2000) Bystander activation of CD4+ T cells accounts for herpetic ocular lesions. Investigative Ophthalmology & Visual Science. 41: 453-9
Gangappa S, Deshpande SP, Rouse BT. (1999) Bystander activation of CD4(+) T cells can represent an exclusive means of immunopathology in a virus infection. European Journal of Immunology. 29: 3674-82
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