Jason A. Wicker, Ph.D.

2007 University of Texas Medical Branch, Galveston, TX 
Microbiology Biology, Molecular Biology, Virology Biology
"Jason Wicker"


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Alan D. T. Barrett grad student 2007 UTMB
 (Mutational analysis of the West Nile virus NS4B protein.)
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Xie G, Whiteman MC, Wicker JA, et al. (2014) In vitro analysis of MyD88-mediated cellular immune response to West Nile virus mutant strain infection. Journal of Visualized Experiments : Jove. e52121
Zhang J, Wang J, Pang L, et al. (2014) The co-stimulatory effects of MyD88-dependent Toll-like receptor signaling on activation of murine γδ T cells. Plos One. 9: e108156
Xie G, Welte T, Wang J, et al. (2013) A West Nile virus NS4B-P38G mutant strain induces adaptive immunity via TLR7-MyD88-dependent and independent signaling pathways. Vaccine. 31: 4143-51
Van Slyke GA, Jia Y, Whiteman MC, et al. (2013) Vertebrate attenuated West Nile virus mutants have differing effects on vector competence in Culex tarsalis mosquitoes. The Journal of General Virology. 94: 1069-72
Wicker JA, Whiteman MC, Beasley DW, et al. (2012) Mutational analysis of the West Nile virus NS4B protein. Virology. 426: 22-33
Whiteman MC, Wicker JA, Kinney RM, et al. (2011) Multiple amino acid changes at the first glycosylation motif in NS1 protein of West Nile virus are necessary for complete attenuation for mouse neuroinvasiveness. Vaccine. 29: 9702-10
Welte T, Xie G, Wicker JA, et al. (2011) Immune responses to an attenuated West Nile virus NS4B-P38G mutant strain. Vaccine. 29: 4853-61
Whiteman MC, Li L, Wicker JA, et al. (2010) Development and characterization of non-glycosylated E and NS1 mutant viruses as a potential candidate vaccine for West Nile virus. Vaccine. 28: 1075-83
Schepp-Berglind J, Luo M, Wang D, et al. (2007) Complex adenovirus-mediated expression of West Nile virus C, PreM, E, and NS1 proteins induces both humoral and cellular immune responses. Clinical and Vaccine Immunology : Cvi. 14: 1117-26
Wicker JA, Whiteman MC, Beasley DW, et al. (2006) A single amino acid substitution in the central portion of the West Nile virus NS4B protein confers a highly attenuated phenotype in mice. Virology. 349: 245-53
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