Nicholas H. Carbonetti

Affiliations: 
University of Maryland School of Medicine, Baltimore, MD, United States 
Area:
Cell Biology, Microbiology Biology
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"Nicholas Carbonetti"
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Publications

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Wang X, Shaw DK, Hammond HL, et al. (2016) The Prostaglandin E2-EP3 Receptor Axis Regulates Anaplasma phagocytophilum-Mediated NLRC4 Inflammasome Activation. Plos Pathogens. 12: e1005803
Plaut RD, Scanlon KM, Taylor M, et al. (2016) Intracellular disassembly and activity of pertussis toxin require interaction with ATP. Pathogens and Disease
Carbonetti NH. (2016) Bordetella pertussis: new concepts in pathogenesis and treatment. Current Opinion in Infectious Diseases
Skerry C, Scanlon K, Rosen H, et al. (2015) Sphingosine-1-phosphate Receptor Agonism Reduces Bordetella pertussis-mediated Lung Pathology. The Journal of Infectious Diseases. 211: 1883-6
Scanlon KM, Gau Y, Zhu J, et al. (2014) Epithelial anion transporter pendrin contributes to inflammatory lung pathology in mouse models of Bordetella pertussis infection. Infection and Immunity. 82: 4212-21
Connelly CE, Sun Y, Carbonetti NH. (2012) Pertussis toxin exacerbates and prolongs airway inflammatory responses during Bordetella pertussis infection. Infection and Immunity. 80: 4317-32
Worthington ZE, Van Rooijen N, Carbonetti NH. (2011) Enhancement of Bordetella parapertussis infection by Bordetella pertussis in mixed infection of the respiratory tract. Fems Immunology and Medical Microbiology. 63: 119-28
Ayala VI, Teijaro JR, Farber DL, et al. (2011) Bordetella pertussis infection exacerbates influenza virus infection through pertussis toxin-mediated suppression of innate immunity. Plos One. 6: e19016
Carbonetti NH. (2010) Pertussis toxin and adenylate cyclase toxin: key virulence factors of Bordetella pertussis and cell biology tools. Future Microbiology. 5: 455-69
Andreasen C, Powell DA, Carbonetti NH. (2009) Pertussis toxin stimulates IL-17 production in response to Bordetella pertussis infection in mice. Plos One. 4: e7079
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