Chao-Hung Lee
Affiliations: | Pathology and Laboratory Medicine | Indiana University, Bloomington, Bloomington, IN, United States |
Area:
Parasitology Biology, Microbiology BiologyGoogle:
"Chao-Hung Lee"
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Publications
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| Zhang C, Lei GS, Shao S, et al. (2012) Accumulation of myeloid-derived suppressor cells in the lungs during Pneumocystis pneumonia. Infection and Immunity. 80: 3634-41 |
| Lasbury ME, Liao CP, Hage CA, et al. (2011) Defective nitric oxide production by alveolar macrophages during Pneumocystis pneumonia. American Journal of Respiratory Cell and Molecular Biology. 44: 540-7 |
| Cheng BH, Liu Y, Xuei X, et al. (2010) Microarray studies on effects of Pneumocystis carinii infection on global gene expression in alveolar macrophages. Bmc Microbiology. 10: 103 |
| Zhang C, Wang SH, Liao CP, et al. (2010) Downregulation of PU.1 leads to decreased expression of Dectin-1 in alveolar macrophages during Pneumocystis pneumonia. Infection and Immunity. 78: 1058-65 |
| Liao CP, Phanstiel O, Lasbury ME, et al. (2009) Polyamine transport as a target for treatment of Pneumocystis pneumonia. Antimicrobial Agents and Chemotherapy. 53: 5259-64 |
| Lasbury ME, Durant PJ, Liao CP, et al. (2009) Effects of decreased calmodulin protein on the survival mechanisms of alveolar macrophages during Pneumocystis pneumonia. Infection and Immunity. 77: 3344-54 |
| Liao CP, Lasbury ME, Wang SH, et al. (2009) Pneumocystis mediates overexpression of antizyme inhibitor resulting in increased polyamine levels and apoptosis in alveolar macrophages. The Journal of Biological Chemistry. 284: 8174-84 |
| Wang SH, Zhang C, Lasbury ME, et al. (2008) Decreased inflammatory response in Toll-like receptor 2 knockout mice is associated with exacerbated Pneumocystis pneumonia. Microbes and Infection / Institut Pasteur. 10: 334-41 |
| Lasbury ME, Wang SH, Zhang C, et al. (2006) Caspase-9 as a target for Pneumocystis pneumonia therapy. The Journal of Eukaryotic Microbiology. 53: S138-9 |
| Lasbury ME, Durant PJ, Tschang D, et al. (2006) Expression and activation of complement protein and alveolar damage during Pneumocystis pneumonia. The Journal of Eukaryotic Microbiology. 53: S136-7 |