David F. Wieczorek
Affiliations: | Molecular Genetics, Biochemistry and Microbiology | University of Cincinnati, Cincinnati, OH |
Area:
Molecular Biology, Genetics, Biochemistry, Physiology BiologyGoogle:
"David Wieczorek"Children
Sign in to add trainee| Kathy K. Pieples | grad student | 2001 | University of Cincinnati |
| Emily M. Schulz | grad student | 2012 | University of Cincinnati |
BETA: Related publications
See more...
Publications
|
You can help our author matching system! If you notice any publications incorrectly attributed to this author, please sign in and mark matches as correct or incorrect. |
| Ryba DM, Warren CM, Karam CN, et al. (2019) Sphingosine-1-Phosphate Receptor Modulator, FTY720, Improves Diastolic Dysfunction and Partially Reverses Atrial Remodeling in a Tm-E180G Mouse Model Linked to Hypertrophic Cardiomyopathy. Circulation. Heart Failure. 12: e005835 |
| Schaefer A, Schneeberger Y, Schulz S, et al. (2019) Analysis of fibrosis in control or pressure overloaded rat hearts after mechanical unloading by heterotopic heart transplantation. Scientific Reports. 9: 5710 |
| Rajan S, Jagatheesan G, Petrashevskaya N, et al. (2018) Tropomyosin pseudo-phosphorylation results in dilated cardiomyopathy. The Journal of Biological Chemistry |
| Alves ML, Warren CM, Simon JN, et al. (2017) Early Sensitization of Myofilaments to Ca2+ Prevents Genetically Linked Dilated Cardiomyopathy in Mice. Cardiovascular Research |
| Wilder T, Ryba DM, Wieczorek DF, et al. (2015) N-acetylcysteine reverses diastolic dysfunction and hypertrophy in familial hypertrophic cardiomyopathy. American Journal of Physiology. Heart and Circulatory Physiology. 309: H1720-30 |
| Tarigopula M, Davis RT, Mungai PT, et al. (2015) Cardiac myosin light chain phosphorylation and inotropic effects of a biased ligand, TRV120023, in a dilated cardiomyopathy model. Cardiovascular Research. 107: 226-34 |
| Prasad V, Lorenz JN, Lasko VM, et al. (2014) Ablation of plasma membrane Ca(2+)-ATPase isoform 4 prevents development of hypertrophy in a model of hypertrophic cardiomyopathy. Journal of Molecular and Cellular Cardiology. 77: 53-63 |
| Simon JN, Chowdhury SA, Warren CM, et al. (2014) Ceramide-mediated depression in cardiomyocyte contractility through PKC activation and modulation of myofilament protein phosphorylation. Basic Research in Cardiology. 109: 445 |
| Alves ML, Dias FA, Gaffin RD, et al. (2014) Desensitization of myofilaments to Ca2+ as a therapeutic target for hypertrophic cardiomyopathy with mutations in thin filament proteins. Circulation. Cardiovascular Genetics. 7: 132-43 |
| Witayavanitkul N, Ait Mou Y, Kuster DW, et al. (2014) Myocardial infarction-induced N-terminal fragment of cardiac myosin-binding protein C (cMyBP-C) impairs myofilament function in human myocardium. The Journal of Biological Chemistry. 289: 8818-27 |