David F. Wieczorek

Molecular Genetics, Biochemistry and Microbiology University of Cincinnati, Cincinnati, OH 
Molecular Biology, Genetics, Biochemistry, Physiology Biology
"David Wieczorek"
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Ryba DM, Warren CM, Karam CN, et al. (2019) Sphingosine-1-Phosphate Receptor Modulator, FTY720, Improves Diastolic Dysfunction and Partially Reverses Atrial Remodeling in a Tm-E180G Mouse Model Linked to Hypertrophic Cardiomyopathy. Circulation. Heart Failure. 12: e005835
Schaefer A, Schneeberger Y, Schulz S, et al. (2019) Analysis of fibrosis in control or pressure overloaded rat hearts after mechanical unloading by heterotopic heart transplantation. Scientific Reports. 9: 5710
Rajan S, Jagatheesan G, Petrashevskaya N, et al. (2018) Tropomyosin pseudo-phosphorylation results in dilated cardiomyopathy. The Journal of Biological Chemistry
Alves ML, Warren CM, Simon JN, et al. (2017) Early Sensitization of Myofilaments to Ca2+ Prevents Genetically Linked Dilated Cardiomyopathy in Mice. Cardiovascular Research
Wilder T, Ryba DM, Wieczorek DF, et al. (2015) N-acetylcysteine reverses diastolic dysfunction and hypertrophy in familial hypertrophic cardiomyopathy. American Journal of Physiology. Heart and Circulatory Physiology. 309: H1720-30
Tarigopula M, Davis RT, Mungai PT, et al. (2015) Cardiac myosin light chain phosphorylation and inotropic effects of a biased ligand, TRV120023, in a dilated cardiomyopathy model. Cardiovascular Research. 107: 226-34
Prasad V, Lorenz JN, Lasko VM, et al. (2014) Ablation of plasma membrane Ca(2+)-ATPase isoform 4 prevents development of hypertrophy in a model of hypertrophic cardiomyopathy. Journal of Molecular and Cellular Cardiology. 77: 53-63
Simon JN, Chowdhury SA, Warren CM, et al. (2014) Ceramide-mediated depression in cardiomyocyte contractility through PKC activation and modulation of myofilament protein phosphorylation. Basic Research in Cardiology. 109: 445
Alves ML, Dias FA, Gaffin RD, et al. (2014) Desensitization of myofilaments to Ca2+ as a therapeutic target for hypertrophic cardiomyopathy with mutations in thin filament proteins. Circulation. Cardiovascular Genetics. 7: 132-43
Witayavanitkul N, Ait Mou Y, Kuster DW, et al. (2014) Myocardial infarction-induced N-terminal fragment of cardiac myosin-binding protein C (cMyBP-C) impairs myofilament function in human myocardium. The Journal of Biological Chemistry. 289: 8818-27
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