Stephen M. Wiesner, Ph.D.

Affiliations: 
2005 University of Minnesota, Twin Cities, Minneapolis, MN 
Area:
Genetics, Molecular Biology
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"Stephen Wiesner"

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David A. Largaespada grad student 2005 UMN
 (RAS pathway activation in the development of myeloproliferative disorders and acute myeloid leukemia.)
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Publications

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Sachs Z, Been RA, DeCoursin KJ, et al. (2016) Stat5 is critical for the development and maintenance of myeloproliferative neoplasm initiated by Nf1 deficiency. Haematologica
Conway-Klaassen JM, Brennecke PJ, Wiesner SM, et al. (2012) Development of online conferencing and web-based in-service modules for preceptor training. Clinical Laboratory Science : Journal of the American Society For Medical Technology. 25: 4-26-33
Conway-Klaassen JM, Wiesner SM, Desens C, et al. (2012) Using online instruction and virtual laboratories to teach hemostasis in a medical laboratory science program. Clinical Laboratory Science : Journal of the American Society For Medical Technology. 25: 224-9
Brueggeman MS, Swinehart C, Yue MJ, et al. (2012) Implementing virtual microscopy improves outcomes in a hematology morphology course. Clinical Laboratory Science : Journal of the American Society For Medical Technology. 25: 149-55
Wiesner SM, Geurts JL, Diers MD, et al. (2011) Nf1 mutant mice with p19ARF gene loss develop accelerated hematopoietic disease resembling acute leukemia with a variable phenotype. American Journal of Hematology. 86: 579-85
Wiesner SM, Decker SA, Larson JD, et al. (2009) De novo induction of genetically engineered brain tumors in mice using plasmid DNA. Cancer Research. 69: 431-9
Wu A, Oh S, Wiesner SM, et al. (2008) Persistence of CD133+ cells in human and mouse glioma cell lines: detailed characterization of GL261 glioma cells with cancer stem cell-like properties. Stem Cells and Development. 17: 173-84
Kim WI, Wiesner SM, Largaespada DA. (2007) Vav promoter-tTA conditional transgene expression system for hematopoietic cells drives high level expression in developing B and T cells. Experimental Hematology. 35: 1231-9
Morgan KJ, Rowley MA, Wiesner SM, et al. (2007) The GAP-related domain of neurofibromin attenuates proliferation and downregulates N- and K-Ras activation in Nf1-negative AML cells. Leukemia Research. 31: 1107-13
Kim A, Morgan K, Hasz DE, et al. (2007) Beta common receptor inactivation attenuates myeloproliferative disease in Nf1 mutant mice. Blood. 109: 1687-91
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