Angela Incassati, Ph.D.
Affiliations: | 2006 | University of Rochester, Rochester, NY |
Area:
Cell BiologyGoogle:
"Angela Incassati"Parents
Sign in to add mentor| Dennis J. McCance | grad student | 2006 | Rochester | |
| (Bypass of cell cycle checkpoints and induction of tetraploidy in primary human epithelial cells by Human Papillomavirus type-16: A role for dysfunctional mitotic regulators.) | ||||
| Pamela Cowin | post-doc | NYU (Cell Biology Tree) | ||
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Publications
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| Spina E, Simundza J, Incassati A, et al. (2022) Gpr125 is a unifying hallmark of multiple mammary progenitors coupled to tumor latency. Nature Communications. 13: 1421 |
| Incassati A, Chandramouli A, Eelkema R, et al. (2010) Key signaling nodes in mammary gland development and cancer: β-catenin. Breast Cancer Research : Bcr. 12: 213 |
| Incassati A, Pinderhughes A, Eelkema R, et al. (2009) Links between transforming growth factor-beta and canonical Wnt signaling yield new insights into breast cancer susceptibility, suppression and tumor heterogeneity. Breast Cancer Research : Bcr. 11: 103 |
| Teissedre B, Pinderhughes A, Incassati A, et al. (2009) MMTV-Wnt1 and -DeltaN89beta-catenin induce canonical signaling in distinct progenitors and differentially activate Hedgehog signaling within mammary tumors. Plos One. 4: e4537 |
| Teissedre B, Pinderhughes A, Incassati A, et al. (2009) Correction: MMTV-Wnt1 and -ΔN89β-Catenin Induce Canonical Signaling in Distinct Progenitors and Differentially Activate Hedgehog Signaling within Mammary Tumors Plos One. 4 |
| Incassati A, Patel D, McCance DJ. (2006) Induction of tetraploidy through loss of p53 and upregulation of Plk1 by human papillomavirus type-16 E6. Oncogene. 25: 2444-51 |
| Patel D, Incassati A, Wang N, et al. (2004) Human papillomavirus type 16 E6 and E7 cause polyploidy in human keratinocytes and up-regulation of G2-M-phase proteins. Cancer Research. 64: 1299-306 |