John C. Scatizzi, Ph.D.
Affiliations: | 2007 | Saint Louis University, St. Louis, MO, United States |
Area:
Immunology, Microbiology Biology, Molecular BiologyGoogle:
"John Scatizzi"Parents
Sign in to add mentor| Harris Perlman | grad student | 2007 | Saint Louis University | |
| (Dual roles for cell cycle inhibitors and apoptotic agonists in macrophages during inflammatory disease.) | ||||
BETA: Related publications
See more...
Publications
|
You can help our author matching system! If you notice any publications incorrectly attributed to this author, please sign in and mark matches as correct or incorrect. |
| Scatizzi JC, Hutcheson J, Pope RM, et al. (2010) Bim-Bcl-2 homology 3 mimetic therapy is effective at suppressing inflammatory arthritis through the activation of myeloid cell apoptosis. Arthritis and Rheumatism. 62: 441-51 |
| Scatizzi JC, Mavers M, Hutcheson J, et al. (2009) The CDK domain of p21 is a suppressor of IL-1beta-mediated inflammation in activated macrophages. European Journal of Immunology. 39: 820-5 |
| Hutcheson J, Scatizzi JC, Siddiqui AM, et al. (2008) Combined deficiency of proapoptotic regulators Bim and Fas results in the early onset of systemic autoimmunity. Immunity. 28: 206-17 |
| Scatizzi JC, Hutcheson J, Bickel E, et al. (2007) Pro-apoptotic Bid is required for the resolution of the effector phase of inflammatory arthritis. Arthritis Research & Therapy. 9: R49 |
| Scatizzi JC, Bickel E, Hutcheson J, et al. (2006) Bim deficiency leads to exacerbation and prolongation of joint inflammation in experimental arthritis. Arthritis and Rheumatism. 54: 3182-93 |
| Woods JM, Klosowska K, Spoden DJ, et al. (2006) A cell-cycle independent role for p21 in regulating synovial fibroblast migration in rheumatoid arthritis. Arthritis Research & Therapy. 8: R113 |
| Scatizzi JC, Hutcheson J, Bickel E, et al. (2006) p21Cip1 is required for the development of monocytes and their response to serum transfer-induced arthritis. The American Journal of Pathology. 168: 1531-41 |
| Hutcheson J, Scatizzi JC, Bickel E, et al. (2005) Combined loss of proapoptotic genes Bak or Bax with Bim synergizes to cause defects in hematopoiesis and in thymocyte apoptosis. The Journal of Experimental Medicine. 201: 1949-60 |
| Brown NJ, Hutcheson J, Bickel E, et al. (2004) Fas death receptor signaling represses monocyte numbers and macrophage activation in vivo. Journal of Immunology (Baltimore, Md. : 1950). 173: 7584-93 |
| Eslick J, Scatizzi JC, Albee L, et al. (2004) IL-4 and IL-10 inhibition of spontaneous monocyte apoptosis is associated with Flip upregulation. Inflammation. 28: 139-45 |