Anel Lizcano
Affiliations: | The University of Texas Health Science Center at San Antonio, San Antonio, TX, United States |
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"Anel Lizcano"
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| Kiser ZM, Lizcano A, Nguyen J, et al. (2019) Decreased erythrocyte binding of Siglec-9 increases neutrophil activation in sickle cell disease. Blood Cells, Molecules & Diseases. 81: 102399 |
| Lizcano A, Secundino I, Döhrmann S, et al. (2017) Erythrocyte sialoglycoproteins engage Siglec-9 on neutrophils to suppress activation. Blood |
| Lizcano A, Suresh Babu RA, Shenoy AT, et al. (2017) Transcriptional organization of pneumococcal psrP-secY2A2 and impact of GtfA and GtfB deletion on PsrP-associated virulence properties. Microbes and Infection |
| Lizcano A, Secundino I, Dohrmann S, et al. (2016) Erythrocyte Sialoglycoproteins Engage Siglec-9 to Maintain Neutrophil Quiescence in the Bloodstream Blood. 128: 1022-1022 |
| Secundino I, Lizcano A, Roupé KM, et al. (2015) Host and pathogen hyaluronan signal through human siglec-9 to suppress neutrophil activation. Journal of Molecular Medicine (Berlin, Germany) |
| Blanchette-Cain K, Hinojosa CA, Akula Suresh Babu R, et al. (2013) Streptococcus pneumoniae biofilm formation is strain dependent, multifactorial, and associated with reduced invasiveness and immunoreactivity during colonization. Mbio. 4: e00745-13 |
| Lizcano A, Sanchez CJ, Orihuela CJ. (2012) A role for glycosylated serine-rich repeat proteins in Gram-positive bacterial pathogenesis Molecular Oral Microbiology. 27: 257-269 |
| Sanchez CJ, Kumar N, Lizcano A, et al. (2011) Streptococcus pneumoniae in biofilms are unable to cause invasive disease due to altered virulence determinant production. Plos One. 6: e28738 |
| Sanchez CJ, Hurtgen BJ, Lizcano A, et al. (2011) Biofilm and planktonic pneumococci demonstrate disparate immunoreactivity to human convalescent sera. Bmc Microbiology. 11: 245 |
| Lizcano A, Chin T, Sauer K, et al. (2010) Early biofilm formation on microtiter plates is not correlated with the invasive disease potential of Streptococcus pneumoniae. Microbial Pathogenesis. 48: 124-30 |