David Reisman

Affiliations: 
University of South Carolina, Columbia, SC 
Area:
gene expression, DNA damage, p53 tumor suppressor
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"David Reisman"

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Bill Sugden grad student 1979-1986 UW Madison
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Publications

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Moxley AH, Reisman D. (2021) Context is key: Understanding the regulation, functional control, and activities of the p53 tumour suppressor. Cell Biochemistry and Function. 39: 235-247
Luo H, Shenoy AK, Li X, et al. (2016) MOF Acetylates the Histone Demethylase LSD1 to Suppress Epithelial-to-Mesenchymal Transition. Cell Reports
Marquez SB, Thompson KW, Lu L, et al. (2014) Beyond Mutations: Additional Mechanisms and Implications of SWI/SNF Complex Inactivation. Frontiers in Oncology. 4: 372
Kahali B, Yu J, Marquez SB, et al. (2014) The silencing of the SWI/SNF subunit and anticancer gene BRM in Rhabdoid tumors. Oncotarget. 5: 3316-32
Polson A, Reisman D. (2014) The bidirectional p53-Wrap53β promoter is controlled by common cis- and trans-regulatory elements. Gene. 538: 138-49
Reisman D, Takahashi P, Polson A, et al. (2012) Transcriptional Regulation of the p53 Tumor Suppressor Gene in S-Phase of the Cell-Cycle and the Cellular Response to DNA Damage. Biochemistry Research International. 2012: 808934
Kothandapani A, Gopalakrishnan K, Kahali B, et al. (2012) Downregulation of SWI/SNF chromatin remodeling factor subunits modulates cisplatin cytotoxicity. Experimental Cell Research. 318: 1973-86
Polson A, Durrett E, Reisman D. (2011) A bidirectional promoter reporter vector for the analysis of the p53/WDR79 dual regulatory element. Plasmid. 66: 169-79
Takahashi P, Polson A, Reisman D. (2011) Elevated transcription of the p53 gene in early S-phase leads to a rapid DNA-damage response during S-phase of the cell cycle. Apoptosis : An International Journal On Programmed Cell Death. 16: 950-8
Polson A, Takahashi P, Reisman D. (2010) ChIP (chromatin immunoprecipitation) analysis demonstrates co-ordinated binding of two transcription factors to the promoter of the p53 tumour-suppressor gene. Cell Biology International. 34: 883-91
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