| Year |
Citation |
Score |
| 2015 |
Durrant DM, Daniels BP, Pasieka T, Dorsey D, Klein RS. CCR5 limits cortical viral loads during West Nile virus infection of the central nervous system. Journal of Neuroinflammation. 12: 233. PMID 26667390 DOI: 10.1186/S12974-015-0447-9 |
0.306 |
|
| 2012 |
Pasieka TJ, Menachery VD, Rosato PC, Leib DA. Corneal replication is an interferon response-independent bottleneck for virulence of herpes simplex virus 1 in the absence of virion host shutoff. Journal of Virology. 86: 7692-5. PMID 22553331 DOI: 10.1128/Jvi.00761-12 |
0.328 |
|
| 2011 |
Pasieka TJ, Cilloniz C, Carter VS, Rosato P, Katze MG, Leib DA. Functional genomics reveals an essential and specific role for Stat1 in protection of the central nervous system following herpes simplex virus corneal infection. Journal of Virology. 85: 12972-81. PMID 21994441 DOI: 10.1128/JVI.06032-11 |
0.319 |
|
| 2010 |
Menachery VD, Pasieka TJ, Leib DA. Interferon regulatory factor 3-dependent pathways are critical for control of herpes simplex virus type 1 central nervous system infection. Journal of Virology. 84: 9685-94. PMID 20660188 DOI: 10.1128/Jvi.00706-10 |
0.309 |
|
| 2009 |
Pasieka TJ, Cilloniz C, Lu B, Teal TH, Proll SC, Katze MG, Leib DA. Host responses to wild-type and attenuated herpes simplex virus infection in the absence of Stat1. Journal of Virology. 83: 2075-87. PMID 19109391 DOI: 10.1128/JVI.02007-08 |
0.349 |
|
| 2008 |
Pasieka TJ, Lu B, Leib DA. Enhanced pathogenesis of an attenuated herpes simplex virus for mice lacking Stat1. Journal of Virology. 82: 6052-5. PMID 18400863 DOI: 10.1128/JVI.00297-08 |
0.403 |
|
| 2008 |
Pasieka TJ, Lu B, Crosby SD, Wylie KM, Morrison LA, Alexander DE, Menachery VD, Leib DA. Herpes simplex virus virion host shutoff attenuates establishment of the antiviral state. Journal of Virology. 82: 5527-35. PMID 18367525 DOI: 10.1128/Jvi.02047-07 |
0.401 |
|
| 2006 |
Pasieka TJ, Baas T, Carter VS, Proll SC, Katze MG, Leib DA. Functional genomic analysis of herpes simplex virus type 1 counteraction of the host innate response. Journal of Virology. 80: 7600-12. PMID 16840339 DOI: 10.1128/Jvi.00333-06 |
0.438 |
|
| 2005 |
Maresova L, Pasieka TJ, Homan E, Gerday E, Grose C. Incorporation of three endocytosed varicella-zoster virus glycoproteins, gE, gH, and gB, into the virion envelope. Journal of Virology. 79: 997-1007. PMID 15613328 DOI: 10.1128/Jvi.79.2.997-1007.2005 |
0.624 |
|
| 2004 |
Pasieka TJ, Maresova L, Shiraki K, Grose C. Regulation of varicella-zoster virus-induced cell-to-cell fusion by the endocytosis-competent glycoproteins gH and gE. Journal of Virology. 78: 2884-96. PMID 14990707 DOI: 10.1128/Jvi.78.6.2884-2896.2004 |
0.598 |
|
| 2003 |
Pasieka TJ, Woolson RF, Grose C. Viral induced fusion and syncytium formation: measurement by the Kolmogorov-Smirnov statistical test. Journal of Virological Methods. 111: 157-61. PMID 12880931 DOI: 10.1016/S0166-0934(03)00152-6 |
0.505 |
|
| 2003 |
Pasieka TJ, Maresova L, Grose C. A functional YNKI motif in the short cytoplasmic tail of varicella-zoster virus glycoprotein gH mediates clathrin-dependent and antibody-independent endocytosis. Journal of Virology. 77: 4191-204. PMID 12634377 DOI: 10.1128/Jvi.77.7.4191-4204.2003 |
0.641 |
|
| 2003 |
Maresova L, Pasieka T, Wagenaar T, Jackson W, Grose C. Identification of the authentic varicella-zoster virus gB (gene 31) initiating methionine overlapping the 3' end of gene 30. Journal of Medical Virology. 70: S64-70. PMID 12627491 DOI: 10.1002/Jmv.10324 |
0.489 |
|
| 2001 |
Maresova L, Pasieka TJ, Grose C. Varicella-zoster Virus gB and gE coexpression, but not gB or gE alone, leads to abundant fusion and syncytium formation equivalent to those from gH and gL coexpression. Journal of Virology. 75: 9483-92. PMID 11533210 DOI: 10.1128/Jvi.75.19.9483-9492.2001 |
0.628 |
|
| 2001 |
Drew PD, Moss MT, Pasieka TJ, Grose C, Harris WJ, Porter AJ. Multimeric humanized varicella-zoster virus antibody fragments to gH neutralize virus while monomeric fragments do not. The Journal of General Virology. 82: 1959-63. PMID 11458003 DOI: 10.1099/0022-1317-82-8-1959 |
0.551 |
|
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