Lauren M. Lilly, Ph.D. - Publications

Affiliations: 
2013 Biology University of Alabama, Birmingham, Birmingham, AL, United States 
Area:
Immunology, Microbiology Biology

6 high-probability publications. We are testing a new system for linking publications to authors. You can help! If you notice any inaccuracies, please sign in and mark papers as correct or incorrect matches. If you identify any major omissions or other inaccuracies in the publication list, please let us know.

Year Citation  Score
2014 Lilly LM, Scopel M, Nelson MP, Burg AR, Dunaway CW, Steele C. Eosinophil deficiency compromises lung defense against Aspergillus fumigatus. Infection and Immunity. 82: 1315-25. PMID 24379296 DOI: 10.1128/IAI.01172-13  0.56
2012 Lilly LM, Gessner MA, Dunaway CW, Metz AE, Schwiebert L, Weaver CT, Brown GD, Steele C. The β-glucan receptor dectin-1 promotes lung immunopathology during fungal allergy via IL-22. Journal of Immunology (Baltimore, Md. : 1950). 189: 3653-60. PMID 22933634 DOI: 10.4049/jimmunol.1201797  0.56
2012 Gessner MA, Werner JL, Lilly LM, Nelson MP, Metz AE, Dunaway CW, Chan YR, Ouyang W, Brown GD, Weaver CT, Steele C. Dectin-1-dependent interleukin-22 contributes to early innate lung defense against Aspergillus fumigatus. Infection and Immunity. 80: 410-7. PMID 22038916 DOI: 10.1128/IAI.05939-11  0.56
2011 Werner JL, Gessner MA, Lilly LM, Nelson MP, Metz AE, Horn D, Dunaway CW, Deshane J, Chaplin DD, Weaver CT, Brown GD, Steele C. Neutrophils produce interleukin 17A (IL-17A) in a dectin-1- and IL-23-dependent manner during invasive fungal infection. Infection and Immunity. 79: 3966-77. PMID 21807912 DOI: 10.1128/IAI.05493-11  0.56
2010 Mackerness KJ, Cox MA, Lilly LM, Weaver CT, Harrington LE, Zajac AJ. Pronounced virus-dependent activation drives exhaustion but sustains IFN-γ transcript levels. Journal of Immunology (Baltimore, Md. : 1950). 185: 3643-51. PMID 20720198 DOI: 10.4049/jimmunol.1000841  0.56
2010 Campbell JA, Davis RS, Lilly LM, Fremont DH, French AR, Carayannopoulos LN. Cutting edge: FcR-like 5 on innate B cells is targeted by a poxvirus MHC class I-like immunoevasin. Journal of Immunology (Baltimore, Md. : 1950). 185: 28-32. PMID 20519648 DOI: 10.4049/jimmunol.1000240  0.56
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