Miriam Samstein, Ph.D. - Publications
Affiliations: | 2014 | Weill Cornell Medical College, New York, NY, United States |
Area:
Immunology, Microbiology BiologyYear | Citation | Score | |||
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2013 | Samstein M, Schreiber HA, Leiner IM, Susac B, Glickman MS, Pamer EG. Essential yet limited role for CCR2⁺ inflammatory monocytes during Mycobacterium tuberculosis-specific T cell priming. Elife. 2: e01086. PMID 24220507 DOI: 10.7554/Elife.01086 | 0.532 | |||
2013 | Samstein M, Schreiber HA, Leiner IM, Sušac B, Glickman MS, Pamer EG. Author response: Essential yet limited role for CCR2+ inflammatory monocytes during Mycobacterium tuberculosis-specific T cell priming Elife. DOI: 10.7554/Elife.01086.013 | 0.525 | |||
2013 | Samstein M, Schreiber HA, Leiner IM, Sušac B, Glickman MS, Pamer EG. Essential yet limited role for CCR2+ inflammatory monocytes during Mycobacterium tuberculosis-specific T cell priming Elife. 2013. DOI: 10.7554/eLife.01086.01086 | 0.52 | |||
2011 | Gallegos AM, van Heijst JW, Samstein M, Su X, Pamer EG, Glickman MS. A gamma interferon independent mechanism of CD4 T cell mediated control of M. tuberculosis infection in vivo. Plos Pathogens. 7: e1002052. PMID 21625591 DOI: 10.1371/Journal.Ppat.1002052 | 0.528 | |||
2010 | Ubeda C, Taur Y, Jenq RR, Equinda MJ, Son T, Samstein M, Viale A, Socci ND, van den Brink MR, Kamboj M, Pamer EG. Vancomycin-resistant Enterococcus domination of intestinal microbiota is enabled by antibiotic treatment in mice and precedes bloodstream invasion in humans. The Journal of Clinical Investigation. 120: 4332-41. PMID 21099116 DOI: 10.1172/Jci43918 | 0.37 | |||
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