☰

Aaron M. Domina, Ph.D. - Publications

Affiliations: 
2001 Dartmouth College, Hanover, NH, United States 
Area:
Pharmacology, Molecular Biology, Cell Biology
Tree Info Grants Similar researchers PubMed Report error

4 high-probability publications. We are testing a new system for linking publications to authors. You can help! If you notice any inaccuracies, please sign in and mark papers as correct or incorrect matches. If you identify any major omissions or other inaccuracies in the publication list, please let us know.

Year Citation  Score
2012 Nifoussi SK, Vrana JA, Domina AM, De Biasio A, Gui J, Gregory MA, Hann SR, Craig RW. Thr 163 phosphorylation causes Mcl-1 stabilization when degradation is independent of the adjacent GSK3-targeted phosphodegron, promoting drug resistance in cancer. Plos One. 7: e47060. PMID 23056582 DOI: 10.1371/Journal.Pone.0047060  0.65
2007 De Biasio A, Vrana JA, Zhou P, Qian L, Bieszczad CK, Braley KE, Domina AM, Weintraub SJ, Neveu JM, Lane WS, Craig RW. N-terminal truncation of antiapoptotic MCL1, but not G2/M-induced phosphorylation, is associated with stabilization and abundant expression in tumor cells. The Journal of Biological Chemistry. 282: 23919-36. PMID 17561513 DOI: 10.1074/Jbc.M700938200  0.623
2004 Domina AM, Vrana JA, Gregory MA, Hann SR, Craig RW. MCL1 is phosphorylated in the PEST region and stabilized upon ERK activation in viable cells, and at additional sites with cytotoxic okadaic acid or taxol. Oncogene. 23: 5301-15. PMID 15241487 DOI: 10.1038/Sj.Onc.1207692  0.646
2000 Domina AM, Smith JH, Craig RW. Myeloid cell leukemia 1 is phosphorylated through two distinct pathways, one associated with extracellular signal-regulated kinase activation and the other with G2/M accumulation or protein phosphatase 1/2A inhibition. The Journal of Biological Chemistry. 275: 21688-94. PMID 10777489 DOI: 10.1074/Jbc.M000915200  0.648
Show low-probability matches.