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Hongyu Zhang, Ph.D.

Affiliations: 
2010 Physiology Temple University, Philadelphia, PA, United States 
Area:
Physiology Biology
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"Hongyu Zhang"
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Steven R. Houser grad student 2010 Temple University
 (Increasing myocyte contractility exacerbates cardiac injury and pump dysfunction and ablation of phosphorylation site in RyR2 at Ser-2808 has no protection against cardiac dysfunction progression after myocardial infarction.)

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Publications

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Wang Y, Zhai W, Zhang H, et al. (2023) Injectable Polyzwitterionic Lubricant for Complete Prevention of Cardiac Adhesion. Macromolecular Bioscience. e2200554
Zhong Y, Zhou Y, Ding R, et al. (2023) Intra-articular treatment of temporomandibular joint osteoarthritis by injecting actively-loaded meloxicam liposomes with dual-functions of anti-inflammation and lubrication. Materials Today. Bio. 19: 100573
Correll RN, Makarewich CA, Zhang H, et al. (2017) Caveolae-localized L-type Ca2+ channels do not contribute to function or hypertrophic signalling in the mouse heart. Cardiovascular Research
Li YF, Huang X, Li X, et al. (2016) Caspase-1 mediates hyperlipidemia-weakened progenitor cell vessel repair. Frontiers in Bioscience (Landmark Edition). 21: 178-91
Zhang X, Ai X, Nakayama H, et al. (2016) Persistent increases in Ca(2+) influx through Cav1.2 shortens action potential and causes Ca(2+) overload-induced afterdepolarizations and arrhythmias. Basic Research in Cardiology. 111: 4
Correll RN, Goonasekera SA, van Berlo JH, et al. (2015) STIM1 elevation in the heart results in aberrant Ca(2+) handling and cardiomyopathy. Journal of Molecular and Cellular Cardiology
Makarewich CA, Zhang H, Davis J, et al. (2014) Transient receptor potential channels contribute to pathological structural and functional remodeling after myocardial infarction. Circulation Research. 115: 567-80
Wang F, Gao H, Kubo H, et al. (2013) T-type Ca²⁺ channels regulate the exit of cardiac myocytes from the cell cycle after birth. Journal of Molecular and Cellular Cardiology. 62: 122-30
Li D, Li H, Ni X, et al. (2013) Construction of a gentamicin C1a-overproducing strain of Micromonospora purpurea by inactivation of the gacD gene. Microbiological Research. 168: 263-7
Gao H, Wang F, Wang W, et al. (2012) Ca(2+) influx through L-type Ca(2+) channels and transient receptor potential channels activates pathological hypertrophy signaling. Journal of Molecular and Cellular Cardiology. 53: 657-67
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