Elizabeth D. Luczak, Ph.D.
Affiliations: | 2007 | Molecular, Cellular and Developmental Biology | University of Colorado, Boulder, Boulder, CO, United States |
Area:
Molecular Biology, Physiology BiologyGoogle:
"Elizabeth Luczak"Parents
Sign in to add mentor| Leslie A. Leinwand | grad student | 2007 | CU Boulder | |
| (Modifiers of cardiac adaptation.) | ||||
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Publications
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| Umapathi P, Banerjee PS, Zachara NE, et al. (2021) Excessive - GlcNAcylation Causes Heart Failure and Sudden Death. Circulation |
| Zhang M, Yang X, Zimmerman RJ, et al. (2020) CaMKII exacerbates heart failure progression by activating class I HDACs. Journal of Molecular and Cellular Cardiology |
| Luczak ED, Wu Y, Granger JM, et al. (2020) Mitochondrial CaMKII causes adverse metabolic reprogramming and dilated cardiomyopathy. Nature Communications. 11: 4416 |
| Konstantinidis K, Bezzerides VJ, Lai L, et al. (2020) MICAL1 constrains cardiac stress responses and protects against disease by oxidizing CaMKII. The Journal of Clinical Investigation |
| Umapathi M, Mesubi O, Granger J, et al. (2019) Abstract 261: Excessive O-GlcNAcylation Causes Heart Failure Circulation Research. 125 |
| Zhang M, Luczak ED, Granger J, et al. (2018) Abstract 457: Ca2+/calmodulin Dependent Kinase II Exacerbates Heart Failure Progression Through Activation of Class I Histone Deacetylases Circulation Research. 123 |
| Luckey SW, Haines CD, Konhilas JP, et al. (2017) Cyclin D2 is a critical mediator of exercise-induced cardiac hypertrophy. Experimental Biology and Medicine (Maywood, N.J.). 1535370217731503 |
| Qu J, Do DC, Zhou Y, et al. (2017) Oxidized CaMKII promotes asthma through the activation of mast cells. Jci Insight. 2: e90139 |
| Mesubi O, Rokita A, Abrol N, et al. (2017) CaMKII oxidation causes increased atrial fibrillation in diabetic mice Journal of Molecular and Cellular Cardiology. 112: 161-162 |
| Wu Y, Rasmussen TP, Koval OM, et al. (2015) The mitochondrial uniporter controls fight or flight heart rate increases. Nature Communications. 6: 6081 |