Melinda C. Myzak, Ph.D.
Affiliations: | 2005 | Oregon State University, Corvallis, OR |
Area:
Molecular Biology, Cell Biology, OncologyGoogle:
"Melinda Myzak"Parents
Sign in to add mentor| Roderick H. Dashwood | grad student | 2005 | Oregon State | |
| (A novel mechanism of chemoprevention by sulforaphane: Inhibition of histone deacetylase.) | ||||
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Publications
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| Myzak MC, Tong P, Dashwood WM, et al. (2007) Sulforaphane retards the growth of human PC-3 xenografts and inhibits HDAC activity in human subjects. Experimental Biology and Medicine (Maywood, N.J.). 232: 227-34 |
| Myzak MC, Ho E, Dashwood RH. (2006) Dietary agents as histone deacetylase inhibitors. Molecular Carcinogenesis. 45: 443-6 |
| Myzak MC, Dashwood RH. (2006) Histone deacetylases as targets for dietary cancer preventive agents: lessons learned with butyrate, diallyl disulfide, and sulforaphane. Current Drug Targets. 7: 443-52 |
| Myzak MC, Dashwood RH. (2006) Chemoprotection by sulforaphane: keep one eye beyond Keap1. Cancer Letters. 233: 208-18 |
| Myzak MC, Dashwood WM, Orner GA, et al. (2006) Sulforaphane inhibits histone deacetylase in vivo and suppresses tumorigenesis in Apc-minus mice. Faseb Journal : Official Publication of the Federation of American Societies For Experimental Biology. 20: 506-8 |
| Myzak MC, Hardin K, Wang R, et al. (2006) Sulforaphane inhibits histone deacetylase activity in BPH-1, LnCaP and PC-3 prostate epithelial cells. Carcinogenesis. 27: 811-9 |
| Dashwood RH, Myzak MC, Ho E. (2006) Dietary HDAC inhibitors: time to rethink weak ligands in cancer chemoprevention? Carcinogenesis. 27: 344-9 |
| Myzak MC, Karplus PA, Chung FL, et al. (2004) A novel mechanism of chemoprotection by sulforaphane: inhibition of histone deacetylase. Cancer Research. 64: 5767-74 |
| Al-Fageeh M, Li Q, Dashwood WM, et al. (2004) Phosphorylation and ubiquitination of oncogenic mutants of beta-catenin containing substitutions at Asp32. Oncogene. 23: 4839-46 |
| Myzak MC, Carr AC. (2002) Myeloperoxidase-dependent caspase-3 activation and apoptosis in HL-60 cells: protection by the antioxidants ascorbate and (dihydro)lipoic acid. Redox Report : Communications in Free Radical Research. 7: 47-53 |