Nuri A. Temiz, Ph.D.
Affiliations: | 2009 | University of Pittsburgh, Pittsburgh, PA, United States |
Area:
Biochemistry, Molecular BiologyGoogle:
"Nuri Temiz"Parents
Sign in to add mentor| Carlos J. Camacho | grad student | 2009 | University of Pittsburgh | |
| (Decoding the protein -DNA recognition rules.) | ||||
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Publications
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| Sanchez A, Ortega P, Sakhtemani R, et al. (2024) Mesoscale DNA features impact APOBEC3A and APOBEC3B deaminase activity and shape tumor mutational landscapes. Nature Communications. 15: 2370 |
| Carpenter MA, Temiz NA, Ibrahim MA, et al. (2023) Mutational impact of APOBEC3A and APOBEC3B in a human cell line and comparisons to breast cancer. Plos Genetics. 19: e1011043 |
| Durfee C, Temiz NA, Levin-Klein R, et al. (2023) Human APOBEC3B promotes tumor development in vivo including signature mutations and metastases. Cell Reports. Medicine. 4: 101211 |
| Sanchez A, Ortega P, Sakhtemani R, et al. (2023) Mesoscale DNA Features Impact APOBEC3A and APOBEC3B Deaminase Activity and Shape Tumor Mutational Landscapes. Biorxiv : the Preprint Server For Biology |
| Naumann JA, Argyris PP, Carpenter MA, et al. (2023) DNA Deamination Is Required for Human APOBEC3A-Driven Hepatocellular Carcinoma In Vivo. International Journal of Molecular Sciences. 24 |
| Law EK, Levin-Klein R, Jarvis MC, et al. (2020) APOBEC3A catalyzes mutation and drives carcinogenesis in vivo. The Journal of Experimental Medicine. 217 |
| Jarvis MC, Ebrahimi D, Temiz NA, et al. (2018) Mutation Signatures Including APOBEC in Cancer Cell Lines. Jnci Cancer Spectrum. 2 |
| Truong TH, Hu H, Temiz NA, et al. (2018) Cancer Stem Cell Phenotypes in ER+ Breast Cancer Models are Promoted by PELP1/AIB1 Complexes. Molecular Cancer Research : McR |
| Scott M, Temiz N, Sarver A, et al. (2018) Abstract PR14: Immune cell transcript levels, metastatic progression, and survival in osteosarcoma: A comparative transcriptome analysis Omics a Journal of Integrative Biology. 24 |
| Sarver AL, Mills L, Temiz N, et al. (2018) Abstract 3399: Comparative genomic analyses of osteosarcoma etiology reveal a chromosomal structural rationale for the increased incidence of osteosarcoma in dogs Cancer Research. 78: 3399-3399 |