Paul M. Carvey, Ph.D.
Affiliations: | Pharmacology | Rush University Medical Center, Chicago, IL, United States |
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| Kousik SM, Napier TC, Ross RD, et al. (2014) Dopamine receptors and the persistent neurovascular dysregulation induced by methamphetamine self-administration in rats. The Journal of Pharmacology and Experimental Therapeutics. 351: 432-9 |
| Kelly LP, Carvey PM, Keshavarzian A, et al. (2014) Progression of intestinal permeability changes and alpha-synuclein expression in a mouse model of Parkinson's disease. Movement Disorders : Official Journal of the Movement Disorder Society. 29: 999-1009 |
| Kousik SM, Carvey PM, Napier TC. (2014) Methamphetamine self-administration results in persistent dopaminergic pathology: implications for Parkinson's disease risk and reward-seeking. The European Journal of Neuroscience. 40: 2707-14 |
| Kousik SM, Napier TC, Carvey PM. (2012) The effects of psychostimulant drugs on blood brain barrier function and neuroinflammation. Frontiers in Pharmacology. 3: 121 |
| Desai Bradaric B, Patel A, Schneider JA, et al. (2012) Evidence for angiogenesis in Parkinson's disease, incidental Lewy body disease, and progressive supranuclear palsy. Journal of Neural Transmission (Vienna, Austria : 1996). 119: 59-71 |
| Kousik SM, Graves SM, Napier TC, et al. (2011) Methamphetamine-induced vascular changes lead to striatal hypoxia and dopamine reduction. Neuroreport. 22: 923-8 |
| Patel A, Toia GV, Colletta K, et al. (2011) An angiogenic inhibitor, cyclic RGDfV, attenuates MPTP-induced dopamine neuron toxicity. Experimental Neurology. 231: 160-70 |
| Monahan J, Ling Z, Carvey P. (2010) Prenatal lipopolysaccharide (LPS) exposure alters the trophic environment of the developing nigro-striatal dopamine (DA) pathway Brain, Behavior, and Immunity. 24: S6-S7 |
| Wang S, Yan JY, Lo YK, et al. (2009) Dopaminergic and serotoninergic deficiencies in young adult rats prenatally exposed to the bacterial lipopolysaccharide. Brain Research. 1265: 196-204 |
| Ling Z, Zhu Y, Tong CW, et al. (2009) Prenatal lipopolysaccharide does not accelerate progressive dopamine neuron loss in the rat as a result of normal aging. Experimental Neurology. 216: 312-20 |