Ian F. Pitha-Rowe, Ph.D.
Affiliations: | 2004 | Dartmouth College, Hanover, NH, United States |
Area:
Molecular BiologyGoogle:
"Ian Pitha-Rowe"Parents
Sign in to add mentor| Ethan Dmitrovsky | grad student | 2004 | Dartmouth | |
| (Post-translational modification in retinoid action: A role for UBE1L in cancer therapy and chemoprevention.) | ||||
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Publications
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| Shah SJ, Blumen S, Pitha-Rowe I, et al. (2008) UBE1L represses PML/RAR{alpha} by targeting the PML domain for ISG15ylation. Molecular Cancer Therapeutics. 7: 905-14 |
| Ma Y, Feng Q, Pitha-Rowe I, et al. (2007) Gene profiling uncovers retinoid target genes. Methods in Molecular Biology (Clifton, N.J.). 383: 101-21 |
| Pitha-Rowe IF, Pitha PM. (2007) Viral defense, carcinogenesis and ISG15: novel roles for an old ISG. Cytokine & Growth Factor Reviews. 18: 409-17 |
| Pitha-Rowe I, Petty WJ, Feng Q, et al. (2004) Microarray analyses uncover UBE1L as a candidate target gene for lung cancer chemoprevention. Cancer Research. 64: 8109-15 |
| Dragnev KH, Pitha-Rowe I, Ma Y, et al. (2004) Specific chemopreventive agents trigger proteasomal degradation of G1 cyclins: implications for combination therapy. Clinical Cancer Research : An Official Journal of the American Association For Cancer Research. 10: 2570-7 |
| Sempere LF, Freemantle S, Pitha-Rowe I, et al. (2004) Expression profiling of mammalian microRNAs uncovers a subset of brain-expressed microRNAs with possible roles in murine and human neuronal differentiation. Genome Biology. 5: R13 |
| Pitha-Rowe I, Hassel BA, Dmitrovsky E. (2004) Involvement of UBE1L in ISG15 conjugation during retinoid-induced differentiation of acute promyelocytic leukemia. The Journal of Biological Chemistry. 279: 18178-87 |
| Pitha-Rowe I, Petty WJ, Kitareewan S, et al. (2003) Retinoid target genes in acute promyelocytic leukemia. Leukemia. 17: 1723-30 |
| Kitareewan S, Pitha-Rowe I, Sekula D, et al. (2002) UBE1L is a retinoid target that triggers PML/RARalpha degradation and apoptosis in acute promyelocytic leukemia. Proceedings of the National Academy of Sciences of the United States of America. 99: 3806-11 |