Miguel A. Pappolla, MD PhD
Affiliations: | 2008- | Neurology | University of Texas Medical Branch, Galveston, TX |
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"Miguel Pappolla"Bio:
http://www.linkedin.com/pub/miguel-pappolla/10/aab/74
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http://www.frontiersin.org/people/miguelpappolla/13139/medicine
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http://www.frontiersin.org/people/miguelpappolla/13139/medicine
Cross-listing: Neurotree - Alzheimer's Tree
Parents
Sign in to add mentor| Samuel M. Chou | grad student | Cleveland Clinic Foundation | |
| Nikolaos K. Robakis | grad student | (Neurotree) | |
| Uros Roessmann | grad student | (Neurotree) | |
| Pierluigi C. Gambetti | post-doc | Case Western | |
| George Perry | post-doc | 1985-1986 | Case Western |
Collaborators
Sign in to add collaborator| Blas Frangione | collaborator | (Neurotree) | |
| Kumar Sambamurti | collaborator | (Neurotree) | |
| Mark Anthony Smith | collaborator | Case Western |
BETA: Related publications
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Publications
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| Poeggeler B, Singh SK, Sambamurti K, et al. (2023) Tryptophan in Nutrition and Health 2.0. International Journal of Molecular Sciences. 24 |
| Pappolla MA, Carare RO, Poeggeler B, et al. (2023) The Lymphatic System in Neurological Disease and Alzheimer's Disease. A Brief Editorial. Current Alzheimer Research. 19: 689-693 |
| Pappolla MA, Perry G, Fang X, et al. (2021) Indoles as essential mediators in the gut-brain axis. Their role in Alzheimer's disease. Neurobiology of Disease. 105403 |
| Pappolla MA, Matsubara E, Vidal R, et al. (2018) Melatonin treatment enhances Aβ lymphatic clearance in a transgenic mouse model of amyloidosis. Current Alzheimer Research |
| Baranello RJ, Bharani KL, Padmaraju V, et al. (2015) Amyloid-beta protein clearance and degradation (ABCD) pathways and their role in Alzheimer's disease. Current Alzheimer Research. 12: 32-46 |
| Baranello RJ, Bharani KL, Padmaraju V, et al. (2015) Amyloid-beta protein clearance and degradation (ABCD) pathways and their role in Alzheimer’s disease Current Alzheimer Research. 12: 32-46 |
| Pappolla M, Sambamurti K, Vidal R, et al. (2014) Evidence for lymphatic Aβ clearance in Alzheimer's transgenic mice. Neurobiology of Disease. 71: 215-9 |
| Barnwell E, Padmaraju V, Baranello R, et al. (2014) Evidence of a novel mechanism for partial γ-secretase inhibition induced paradoxical increase in secreted amyloid β protein. Plos One. 9: e91531 |
| Pappolla M, Sambamurti K, Vidal R, et al. (2014) Evidence for lymphatic Aβ clearance in Alzheimer's transgenic mice Neurobiology of Disease. 71: 215-219 |
| Pinnix I, Ghiso JA, Pappolla MA, et al. (2013) Major carboxyl terminal fragments generated by γ-secretase processing of the Alzheimer amyloid precursor are 50 and 51 amino acids long. The American Journal of Geriatric Psychiatry : Official Journal of the American Association For Geriatric Psychiatry. 21: 474-83 |