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David A. Prince

Affiliations: 
Stanford University, Palo Alto, CA 
Area:
Epilepsy
Website:
http://med.stanford.edu/profiles/David_Prince/
Google:
"David Prince"
Cross-listing: Neurotree

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Parents

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Children

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Yunxiang Chu research assistant 2004-2008 Stanford (Neurotree)
Michael J. Gutnick grad student 1968-1972 Stanford (Neurotree)
Jeffrey L. Noebels grad student 1973-1977 Stanford Medical School (Neurotree)
Scott M. Thompson grad student 1986 Stanford (Neurotree)
Alberto Bacci post-doc European Brain Research Institute (Neurotree)
Tom Brown post-doc Stanford Medical School (Neurotree)
Douglas A. Coulter post-doc Stanford (Neurotree)
Robert Fisher post-doc Johns Hopkins (Neurotree)
Kevin D. Graber post-doc Stanford (Neurotree)
John R. Huguenard post-doc Stanford (Neurotree)
Xiaoming Jin post-doc Stanford
Arnold R. Kriegstein post-doc UCSF (Neurotree)
David A. McCormick post-doc Stanford (Neurotree)
Timothy A. Pedley post-doc Columbia (Neurotree)
Qian-Quan Sun post-doc Stanford (Neurotree)
Robert KS Wong post-doc Stanford (Neurotree)
D. Koji Takahashi post-doc 2009- University of Utah (Neurotree)
Feng Gu post-doc 2013- Stanford (Neurotree)
Phillip A. Schwartzkroin post-doc 1978 Stanford
Barry W. Connors post-doc 1980-1982 Stanford (Neurotree)
Heiko J. Luhmann post-doc 1987-1990 Stanford University Medical School (Neurotree)
Charles L. Cox post-doc 1993-1996 Stanford (Neurotree)
Kimberle M. Jacobs post-doc 2001 Stanford (Neurotree)
Viktor N. Kharazia post-doc 1997-2002 Stanford Medical School (Neurotree)
Anita E. Bandrowski post-doc 2000-2004 Stanford University Medical Center (Neurotree)
Sonal Goswami post-doc 2014-2015 Stanford Medical School (Neurotree)

Collaborators

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Irving Wagman collaborator UC Davis (Neurotree)
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Publications

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Bagley JR, Tan Y, Zhu W, et al. (2023) Neuron Navigator 1 (Nav1) regulates the response to cocaine in mice. Communications Biology. 6: 1053
Gu F, Parada I, Yang T, et al. (2022) Chronic partial TrkB activation reduces seizures and mortality in a mouse model of Dravet syndrome. Proceedings of the National Academy of Sciences of the United States of America. 119
Gu F, Parada I, Yang T, et al. (2020) Partial Activation of TrkB Receptors Corrects Interneuronal Calcium Channel Dysfunction and Reduces Epileptogenic Activity in Neocortex following Injury. Cerebral Cortex (New York, N.Y. : 1991)
Perez-Ramirez MB, Gu F, Prince DA. (2020) Prolonged prophylactic effects of gabapentin on status epilepticus-induced neocortical injury. Neurobiology of Disease. 104949
Klein P, Friedman A, Hameed MQ, et al. (2020) Repurposed molecules for antiepileptogenesis: Missing an opportunity to prevent epilepsy? Epilepsia. 61: 359-386
Gu F, Parada I, Yang T, et al. (2018) Partial TrkB receptor activation suppresses cortical epileptogenesis through actions on parvalbumin interneurons. Neurobiology of Disease
Takahashi DK, Jin S, Prince DA. (2017) Gabapentin Prevents Progressive Increases in Excitatory Connectivity and Epileptogenesis Following Neocortical Trauma. Cerebral Cortex (New York, N.Y. : 1991). 1-16
Gu F, Parada I, Shen F, et al. (2017) Structural alterations in fast-spiking GABAergic interneurons in a model of posttraumatic neocortical epileptogenesis. Neurobiology of Disease
Kim SY, Senatorov VV, Morrissey CS, et al. (2017) TGFβ signaling is associated with changes in inflammatory gene expression and perineuronal net degradation around inhibitory neurons following various neurological insults. Scientific Reports. 7: 7711
Faria LC, Gu F, Parada I, et al. (2017) Epileptiform activity and behavioral arrests in mice overexpressing the calcium channel subunit α2δ-1. Neurobiology of Disease
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