Year |
Citation |
Score |
2013 |
Nallagatla SR, Jones CN, Ghosh SK, Sharma SD, Cameron CE, Spremulli LL, Bevilacqua PC. Native tertiary structure and nucleoside modifications suppress tRNA's intrinsic ability to activate the innate immune sensor PKR. Plos One. 8: e57905. PMID 23483938 DOI: 10.1371/Journal.Pone.0057905 |
0.593 |
|
2011 |
Bilbille Y, Gustilo EM, Harris KA, Jones CN, Lusic H, Kaiser RJ, Delaney MO, Spremulli LL, Deiters A, Agris PF. The human mitochondrial tRNAMet: structure/function relationship of a unique modification in the decoding of unconventional codons. Journal of Molecular Biology. 406: 257-74. PMID 21168417 DOI: 10.1016/J.Jmb.2010.11.042 |
0.636 |
|
2010 |
Akama K, Christian BE, Jones CN, Ueda T, Takeuchi N, Spremulli LL. Analysis of the functional consequences of lethal mutations in mitochondrial translational elongation factors. Biochimica Et Biophysica Acta. 1802: 692-8. PMID 20435138 DOI: 10.1016/J.Bbadis.2010.04.003 |
0.585 |
|
2009 |
Jones CN, Miller C, Tenenbaum A, Spremulli LL, Saada A. Antibiotic effects on mitochondrial translation and in patients with mitochondrial translational defects. Mitochondrion. 9: 429-37. PMID 19671450 DOI: 10.1016/J.Mito.2009.08.001 |
0.66 |
|
2008 |
Jones CN, Jones CI, Graham WD, Agris PF, Spremulli LL. A disease-causing point mutation in human mitochondrial tRNAMet rsults in tRNA misfolding leading to defects in translational initiation and elongation. The Journal of Biological Chemistry. 283: 34445-56. PMID 18835817 DOI: 10.1074/Jbc.M806992200 |
0.716 |
|
2008 |
Jones CN, Wilkinson KA, Hung KT, Weeks KM, Spremulli LL. Lack of secondary structure characterizes the 5' ends of mammalian mitochondrial mRNAs. Rna (New York, N.Y.). 14: 862-71. PMID 18367717 DOI: 10.1261/Rna.909208 |
0.64 |
|
2007 |
Jones CN, Spremulli LL, Agris PF. Structural and biochemical investigation of disease causing mutations in human mitochondrial tRNA Met The Faseb Journal. 21. DOI: 10.1096/Fasebj.21.5.A279-B |
0.683 |
|
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