| Year |
Citation |
Score |
| 2022 |
Xolalpa-Cueva L, García-Carlos CA, Villaseñor-Zepeda R, Orta-Salazar E, Díaz-Cintra S, Peña-Ortega F, Perry G, Mondragón-Rodríguez S. Hyperphosphorylated Tau Relates to Improved Cognitive Performance and Reduced Hippocampal Excitability in the Young rTg4510 Mouse Model of Tauopathy. Journal of Alzheimer's Disease : Jad. PMID 35342085 DOI: 10.3233/JAD-215186 |
0.558 |
|
| 2020 |
Mondragón-Rodríguez S, Ordaz B, Orta-Salazar E, Díaz-Cintra S, Peña-Ortega F, Perry G. Hippocampal Unicellular Recordings and Hippocampal-dependent Innate Behaviors in an Adolescent Mouse Model of Alzheimer's disease. Bio-Protocol. 10: e3529. PMID 33654753 DOI: 10.21769/BioProtoc.3529 |
0.468 |
|
| 2020 |
Mondragón-Rodríguez S, Salgado-Burgos H, Peña-Ortega F. Circuitry and Synaptic Dysfunction in Alzheimer's Disease: A New Tau Hypothesis. Neural Plasticity. 2020: 2960343. PMID 32952546 DOI: 10.1155/2020/2960343 |
0.595 |
|
| 2018 |
Mondragón-Rodríguez S, Gu N, Fasano C, Peña-Ortega F, Williams S. dFunctional connectivity between Hippocampus and Lateral septum is affected in very young Alzheimer's transgenic mouse model. Neuroscience. PMID 30599271 DOI: 10.1016/J.Neuroscience.2018.12.042 |
0.45 |
|
| 2018 |
Mondragón-Rodríguez S, Gu N, Manseau F, Williams S. Alzheimer's Transgenic Model Is Characterized by Very Early Brain Network Alterations and β-CTF Fragment Accumulation: Reversal by β-Secretase Inhibition. Frontiers in Cellular Neuroscience. 12: 121. PMID 29867356 DOI: 10.3389/Fncel.2018.00121 |
0.444 |
|
| 2016 |
Mahar I, Albuquerque MS, Mondragon-Rodriguez S, Cavanagh C, Davoli MA, Chabot JG, Williams S, Mechawar N, Quirion R, Krantic S. Phenotypic Alterations in Hippocampal NPY- and PV-Expressing Interneurons in a Presymptomatic Transgenic Mouse Model of Alzheimer's Disease. Frontiers in Aging Neuroscience. 8: 327. PMID 28154533 DOI: 10.3389/Fnagi.2016.00327 |
0.495 |
|
| 2016 |
Mondragón-Rodríguez S, Perry G, Peña-Ortega F, Williams S. Tau, Amyloid beta and Deep brain stimulation: Aiming to restore cognitive deficit in Alzheimer's disease. Current Alzheimer Research. PMID 27539594 DOI: 10.2174/1567205013666160819131336 |
0.671 |
|
| 2015 |
Zhang F, Su B, Wang C, Siedlak SL, Mondragon-Rodriguez S, Lee HG, Wang X, Perry G, Zhu X. Posttranslational modifications of α-tubulin in alzheimer disease. Translational Neurodegeneration. 4: 9. PMID 26029362 DOI: 10.1186/S40035-015-0030-4 |
0.651 |
|
| 2012 |
Mondragón-Rodríguez S, Perry G, Zhu X, Moreira PI, Williams S. Glycogen synthase kinase 3: a point of integration in Alzheimer's disease and a therapeutic target? International Journal of Alzheimer's Disease. 2012: 276803. PMID 22779025 DOI: 10.1155/2012/276803 |
0.646 |
|
| 2012 |
Mondragón-Rodríguez S, Perry G, Zhu X, Boehm J. Amyloid Beta and tau proteins as therapeutic targets for Alzheimer's disease treatment: rethinking the current strategy. International Journal of Alzheimer's Disease. 2012: 630182. PMID 22482074 DOI: 10.1155/2012/630182 |
0.701 |
|
| 2011 |
Mondragon-Rodriguez S, Burgeois C, Boehm J. Tau phosphorylation links Amyloid beta and NMDA receptor activation, implications for Alzheimer's disease Alzheimers & Dementia. 7. DOI: 10.1016/J.Jalz.2011.09.068 |
0.49 |
|
| 2011 |
Collins GH, Mondragón-Rodríguez S, Nunomura A, Zhu X, Moreira PI, Smith MA. Oxidative Stress and Balance in Neurodegenerative Diseases Neurodegeneration: the Molecular Pathology of Dementia and Movement Disorders: Second Edition. 10-12. DOI: 10.1002/9781444341256.ch3 |
0.57 |
|
| 2009 |
Mondragón-Rodríguez S, Basurto-Islas G, Binder LI, García-Sierra F. Conformational changes and cleavage; are these responsible for the tau aggregation in Alzheimer's disease? Future Neurology. 4: 39-53. DOI: 10.2217/14796708.4.1.39 |
0.679 |
|
| Show low-probability matches. |