☰

Sukwon Lee, Ph.D. - Publications

Affiliations: 
Seoul National University, Seoul, South Korea 
 Department of Neural Disease Korea Brain Research Institute 
Area:
fear memory modulation
Tree Info Grants Similar researchers PubMed Report error

26 high-probability publications. We are testing a new system for linking publications to authors. You can help! If you notice any inaccuracies, please sign in and mark papers as correct or incorrect matches. If you identify any major omissions or other inaccuracies in the publication list, please let us know.

Year Citation  Score
2020 Song S, Kim J, Park K, Lee J, Park S, Lee S, Kim J, Hong I, Song B, Choi S. GSK-3β activation is required for ZIP-induced disruption of learned fear. Scientific Reports. 10: 18227. PMID 33106552 DOI: 10.1038/s41598-020-75130-5  0.786
2020 Joo B, Koo JW, Lee S. Posterior parietal cortex mediates fear renewal in a novel context. Molecular Brain. 13: 16. PMID 32024548 DOI: 10.1186/S13041-020-0556-Y  0.321
2018 Lee S, Kim J, Choi S. Endogenous amyloid-β mediates memory forgetting in the normal brain. Biochemical and Biophysical Research Communications. PMID 30361088 DOI: 10.1016/J.Bbrc.2018.10.118  0.792
2017 An B, Kim J, Park K, Lee S, Song S, Choi S. Amount of fear extinction changes its underlying mechanisms. Elife. 6. PMID 28671550 DOI: 10.7554/Elife.25224  0.779
2017 An B, Kim J, Park K, Lee S, Song S, Choi S. Author response: Amount of fear extinction changes its underlying mechanisms Elife. DOI: 10.7554/Elife.25224.018  0.79
2016 Park S, Lee J, Park K, Kim J, Song B, Hong I, Kim J, Lee S, Choi S. Sound tuning of amygdala plasticity in auditory fear conditioning. Scientific Reports. 6: 31069. PMID 27488731 DOI: 10.1038/Srep31069  0.808
2015 Kim J, An B, Kim J, Park S, Park S, Hong I, Lee S, Park K, Choi S. mGluR2/3 in the Lateral Amygdala is Required for Fear Extinction: Cortical Input Synapses onto the Lateral Amygdala as a Target Site of the mGluR2/3 Action. Neuropsychopharmacology : Official Publication of the American College of Neuropsychopharmacology. PMID 26081171 DOI: 10.1038/Npp.2015.145  0.822
2014 Park K, Song S, Hong I, Song B, Kim J, Park S, Lee J, Song S, An B, Kim J, Lee CJ, Shin KS, Choi S, Lee S. Group I mGluR-dependent depotentiation in the lateral amygdala does not require the removal of calcium-permeable AMPA receptors. Frontiers in Behavioral Neuroscience. 8: 269. PMID 25152720 DOI: 10.3389/Fnbeh.2014.00269  0.796
2014 Park K, Song B, Kim J, Hong I, Song S, Lee J, Park S, Kim J, An B, Lee HW, Lee S, Kim H, Lee JC, Lee S, Choi S. ABA renewal involves enhancements in both GluA2-lacking AMPA receptor activity and GluA1 phosphorylation in the lateral amygdala. Plos One. 9: e100108. PMID 24925360 DOI: 10.1371/Journal.Pone.0100108  0.796
2013 Lee S, Song B, Kim J, Park K, Hong I, An B, Song S, Lee J, Park S, Kim J, Park D, Lee CJ, Kim K, Shin KS, Tsien RW, et al. GluA1 phosphorylation at serine 831 in the lateral amygdala is required for fear renewal. Nature Neuroscience. 16: 1436-44. PMID 23974710 DOI: 10.1038/Nn.3491  0.815
2013 Hong I, Kim J, Kim J, Lee S, Ko HG, Nader K, Kaang BK, Tsien RW, Choi S. AMPA receptor exchange underlies transient memory destabilization on retrieval. Proceedings of the National Academy of Sciences of the United States of America. 110: 8218-23. PMID 23630279 DOI: 10.1073/Pnas.1305235110  0.837
2013 Hong I, Kang T, Yoo Y, Park R, Lee J, Lee S, Kim J, Song B, Kim SY, Moon M, Yun KN, Kim JY, Mook-Jung I, Park YM, Choi S. Quantitative proteomic analysis of the hippocampus in the 5XFAD mouse model at early stages of Alzheimer's disease pathology. Journal of Alzheimer's Disease : Jad. 36: 321-34. PMID 23603400 DOI: 10.3233/Jad-130311  0.767
2013 Hong I, Kang T, Yun KN, Yoo Y, Park S, Kim J, An B, Song S, Lee S, Kim J, Song B, Kwon KH, Kim JY, Park YM, Choi S. Quantitative proteomics of auditory fear conditioning. Biochemical and Biophysical Research Communications. 434: 87-94. PMID 23542466 DOI: 10.1016/J.Bbrc.2013.03.060  0.807
2012 Song B, Lee S, Choi S. LY404187, a potentiator of AMPARs, enhances both the amplitude and 1/CV2 of AMPA EPSCs but not NMDA EPSCs at CA3-CA1 synapses in the hippocampus of neonatal rats. Neuroscience Letters. 531: 193-7. PMID 23103715 DOI: 10.1016/J.Neulet.2012.10.020  0.815
2012 Park S, Lee S, Kim J, Choi S. Ex vivo depotentiation of conditioning-induced potentiation at thalamic input synapses onto the lateral amygdala requires GluN2B-containing NMDA receptors. Neuroscience Letters. 530: 121-6. PMID 23069667 DOI: 10.1016/J.Neulet.2012.10.011  0.804
2012 Hong I, Kim J, Song B, Park K, Shin K, Eom KD, Han PL, Lee S, Choi S. Fear conditioning occludes late-phase long-term potentiation at thalamic input synapses onto the lateral amygdala in rat brain slices. Neuroscience Letters. 506: 121-5. PMID 22079527 DOI: 10.1016/J.Neulet.2011.10.063  0.84
2011 Hong I, Kim J, Lee J, Park S, Song B, Kim J, An B, Park K, Lee HW, Lee S, Kim H, Park SH, Eom KD, Lee S, Choi S. Reversible plasticity of fear memory-encoding amygdala synaptic circuits even after fear memory consolidation. Plos One. 6: e24260. PMID 21949700 DOI: 10.1371/Journal.Pone.0024260  0.824
2011 Lee EJ, Son GH, Chung S, Lee S, Kim J, Choi S, Kim K. Impairment of fear memory consolidation in maternally stressed male mouse offspring: evidence for nongenomic glucocorticoid action on the amygdala. The Journal of Neuroscience : the Official Journal of the Society For Neuroscience. 31: 7131-40. PMID 21562275 DOI: 10.1523/Jneurosci.4692-10.2011  0.836
2011 Hong I, Kim J, Song B, Park S, Lee J, Kim J, An B, Lee S, Choi S. Modulation of fear memory by retrieval and extinction: a clue for memory deconsolidation. Reviews in the Neurosciences. 22: 205-29. PMID 21476941 DOI: 10.1515/Rns.2011.023  0.782
2011 Lee S, Kim J, Choi S. In vitro synaptic reconsolidation in amygdala slices prepared from rat brains. Biochemical and Biophysical Research Communications. 407: 339-42. PMID 21385565 DOI: 10.1016/J.Bbrc.2011.03.019  0.854
2010 Kim J, Song B, Hong I, Kim J, Lee J, Park S, Eom JY, Lee CJ, Lee S, Choi S. Reactivation of fear memory renders consolidated amygdala synapses labile. The Journal of Neuroscience : the Official Journal of the Society For Neuroscience. 30: 9631-40. PMID 20631192 DOI: 10.1523/Jneurosci.0940-10.2010  0.832
2009 Hong I, Song B, Lee S, Kim J, Kim J, Choi S. Extinction of cued fear memory involves a distinct form of depotentiation at cortical input synapses onto the lateral amygdala. The European Journal of Neuroscience. 30: 2089-99. PMID 20128847 DOI: 10.1111/J.1460-9568.2009.07004.X  0.854
2009 Kim J, Park S, Lee S, Choi S. Amygdala depotentiation ex vivo requires mitogen-activated protein kinases and protein synthesis. Neuroreport. 20: 517-20. PMID 19247210 DOI: 10.1097/Wnr.0B013E328329412D  0.83
2007 Kim J, Lee S, Park K, Hong I, Song B, Son G, Park H, Kim WR, Park E, Choe HK, Kim H, Lee C, Sun W, Kim K, Shin KS, et al. Amygdala depotentiation and fear extinction. Proceedings of the National Academy of Sciences of the United States of America. 104: 20955-60. PMID 18165656 DOI: 10.1073/Pnas.0710548105  0.856
2007 Park K, Lee S, Kang SJ, Choi S, Shin KS. Hyperpolarization-activated currents control the excitability of principal neurons in the basolateral amygdala. Biochemical and Biophysical Research Communications. 361: 718-24. PMID 17678627 DOI: 10.1016/J.Bbrc.2007.07.064  0.721
2007 Kim J, Lee S, Park H, Song B, Hong I, Geum D, Shin K, Choi S. Blockade of amygdala metabotropic glutamate receptor subtype 1 impairs fear extinction. Biochemical and Biophysical Research Communications. 355: 188-93. PMID 17292864 DOI: 10.1016/J.Bbrc.2007.01.125  0.839
Show low-probability matches.