| Year |
Citation |
Score |
| 2023 |
Aboulesia R, Devilée L, Miller J, Reid J, Salama AB, Ou Q, Jamal M, Nong Y, Andres D, Satin J, Mohamed T, Hudson J. Pharmacological or genetic inhibition of LTCC promotes cardiomyocyte proliferation through inhibition of calcineurin activity. Research Square. PMID 38076903 DOI: 10.21203/rs.3.rs-3552794/v1 |
0.338 |
|
| 2023 |
DeHaan RL, Fujii S, Satin J. Cell Interactions in Cardiac Development: (induction/local cues/heart development/heartrate/ion channels). Development, Growth & Differentiation. 32: 233-241. PMID 37281576 DOI: 10.1111/j.1440-169X.1990.00233.x |
0.372 |
|
| 2021 |
Ahern BM, Sebastian A, Levitan BM, Goh J, Andres DA, Satin J. L-type channel inactivation balances the increased peak calcium current due to absence of Rad in cardiomyocytes. The Journal of General Physiology. 153. PMID 34269819 DOI: 10.1085/jgp.202012854 |
0.315 |
|
| 2020 |
Ahern B, Sebastian A, Andres DA, Satin J. Myocardial RAD Deletion Increases Early L-type Calcium Current without Affecting Late Calcium Current through Multiple Mechanisms Biophysical Journal. 118: 105a. DOI: 10.1016/J.Bpj.2019.11.726 |
0.379 |
|
| 2019 |
Ahern BM, Levitan BM, Veeranki S, Shah M, Ali N, Sebastian A, Su W, Gong MC, Li J, Stelzer JE, Andres DA, Satin J. Myocardial-restricted ablation of the GTPase RAD results in a pro-adaptive heart response in mice. The Journal of Biological Chemistry. PMID 31147441 DOI: 10.1074/Jbc.Ra119.008782 |
0.477 |
|
| 2019 |
Ahern BM, Satin J. The L-type calcium channel current modulation mechanism: the plot thickens and fogs. The Journal of Clinical Investigation. 129: 496-498. PMID 30614816 DOI: 10.1172/Jci125958 |
0.453 |
|
| 2019 |
Ahern B, Levitan B, Veeranki S, Shah M, Ali N, Sebastian A, Li J, Stelzer JE, Andres D, Satin J. Abstract 807: Rad Ablation as a Treatment to Target Cardiac Inotropy via L-type Calcium Channel Function Circulation Research. 125. DOI: 10.1161/Res.125.Suppl_1.807 |
0.464 |
|
| 2019 |
Ahern B, Shah M, Sebastian A, Andres DA, Satin J. Myocardial Rad Deletion Modulates L-Type Calcium Channel Current Biophysical Journal. 116: 237a. DOI: 10.1016/J.Bpj.2018.11.1302 |
0.485 |
|
| 2018 |
Manning JR, Chelvarajan L, Levitan BM, Withers CN, Nagareddy PR, Haggerty CM, Fornwalt BK, Gao E, Tripathi H, Abdel-Latif A, Andres DA, Satin J. Rad GTPase deletion attenuates post-ischemic cardiac dysfunction and remodeling. Jacc. Basic to Translational Science. 3: 83-96. PMID 29732439 DOI: 10.1016/J.Jacbts.2017.09.004 |
0.376 |
|
| 2016 |
Levitan BM, Manning JR, Withers CN, Smith JD, Shaw RM, Andres DA, Sorrell VL, Satin J. Rad-deletion Phenocopies Tonic Sympathetic Stimulation of the Heart. Journal of Cardiovascular Translational Research. PMID 27798760 DOI: 10.1007/S12265-016-9716-Y |
0.369 |
|
| 2015 |
Manning JR, Withers CN, Levitan B, Smith JD, Andres DA, Satin J. Loss of Rad-GTPase Produces a Novel Adaptive Cardiac Phenotype Resistant to Systolic decline with Aging. American Journal of Physiology. Heart and Circulatory Physiology. ajpheart.00389.2015. PMID 26371164 DOI: 10.1152/Ajpheart.00389.2015 |
0.434 |
|
| 2015 |
Magyar J, Banyasz T, Szentandrassy N, Kistamas K, Nanasi PP, Satin J. Role of gap junction channel in the development of beat-to-beat action potential repolarization variability and arrhythmias. Current Pharmaceutical Design. 21: 1042-52. PMID 25354184 DOI: 10.2174/1381612820666141029102443 |
0.463 |
|
| 2014 |
Yin G, Hassan F, Haroun AR, Murphy LL, Crotti L, Schwartz PJ, George AL, Satin J. Arrhythmogenic calmodulin mutations disrupt intracellular cardiomyocyte Ca2+ regulation by distinct mechanisms. Journal of the American Heart Association. 3: e000996. PMID 24958779 DOI: 10.1161/Jaha.114.000996 |
0.342 |
|
| 2013 |
Manning JR, Yin G, Kaminski CN, Magyar J, Feng HZ, Penn J, Sievert G, Thompson K, Jin JP, Andres DA, Satin J. Rad GTPase deletion increases L-type calcium channel current leading to increased cardiac contraction. Journal of the American Heart Association. 2: e000459. PMID 24334906 DOI: 10.1161/Jaha.113.000459 |
0.52 |
|
| 2013 |
Satin J. The long and short of PKC modulation of the L-type calcium channel. Channels (Austin, Tex.). 7: 57-8. PMID 23665613 DOI: 10.4161/Chan.24147 |
0.487 |
|
| 2013 |
Crump SM, Andres DA, Sievert G, Satin J. The cardiac L-type calcium channel distal carboxy terminus autoinhibition is regulated by calcium. American Journal of Physiology. Heart and Circulatory Physiology. 304: H455-64. PMID 23203963 DOI: 10.1152/Ajpheart.00396.2012 |
0.524 |
|
| 2012 |
Magyar J, Kiper CE, Sievert G, Cai W, Shi GX, Crump SM, Li L, Niederer S, Smith N, Andres DA, Satin J. Rem-GTPase regulates cardiac myocyte L-type calcium current. Channels (Austin, Tex.). 6: 166-73. PMID 22854599 DOI: 10.4161/Chan.20192 |
0.51 |
|
| 2012 |
Crump SM, Satin J. The Cardiac L-Type Calcium Channel Distal Carboxyl Terminus is a Reverse use Dependent Inhibitor of Ca Current in Cardiomyocytes Biophysical Journal. 102. DOI: 10.1016/J.Bpj.2011.11.702 |
0.553 |
|
| 2011 |
Magyar J, Jenes A, Kistamas K, Ruzsnavszky F, Nanasi PP, Satin J, Szentandrassy N, Banyasz T. Long term regulation of cardiac L-type calcium channel by small G proteins Current Medicinal Chemistry. 18: 3714-3719. PMID 21774757 DOI: 10.2174/092986711796642436 |
0.539 |
|
| 2011 |
Satin J, Schroder EA, Crump SM. L-type calcium channel auto-regulation of transcription. Cell Calcium. 49: 306-13. PMID 21295347 DOI: 10.1016/J.Ceca.2011.01.001 |
0.459 |
|
| 2011 |
Crump S, Satin J. The Cardiac L-Type Calcium Channel (LTCC) Distal Carboxyl-Terminus (DCT) is a Reverse-Use Dependent Inhibitor of Ca-Current in Cardiomyocytes That Dynamically Increases The Range of Frequency-Dependent Responsiveness Heart Rhythm. 8: 1823. DOI: 10.1016/J.HRTHM.2011.09.039 |
0.469 |
|
| 2011 |
Crump SM, Schroder EA, Sievert GA, Andres DA, Satin J. Calmodulin Interferes with Cav1.2 C-Terminal Regulation of L-Type Channel Current Biophysical Journal. 100: 529a. DOI: 10.1016/J.Bpj.2010.12.3090 |
0.508 |
|
| 2010 |
Pang C, Crump SM, Jin L, Correll RN, Finlin BS, Satin J, Andres DA. Rem GTPase interacts with the proximal CaV1.2 C-terminus and modulates calcium-dependent channel inactivation. Channels (Austin, Tex.). 4: 192-202. PMID 20458179 DOI: 10.4161/Chan.4.3.11867 |
0.565 |
|
| 2010 |
Crump SM, Byse MJ, Andres DA, Satin J. The L-Type Calcium Channel C-Terminus is a Mobile Domain that Competes with Calmodulin Modulation of Calcium Current Biophysical Journal. 98: 518a-519a. DOI: 10.1016/J.Bpj.2009.12.2820 |
0.504 |
|
| 2009 |
Satin J, Schroder EA. Autoregulation of cardiac l-type calcium channels. Trends in Cardiovascular Medicine. 19: 268-71. PMID 20447569 DOI: 10.1016/J.Tcm.2010.02.009 |
0.466 |
|
| 2009 |
Schroder E, Byse M, Satin J. L-type calcium channel C terminus autoregulates transcription. Circulation Research. 104: 1373-81. PMID 19461046 DOI: 10.1161/Circresaha.108.191387 |
0.512 |
|
| 2008 |
Lester WC, Schroder EA, Burgess DE, Yozwiak D, Andres DA, Satin J. Steady-state coupling of plasma membrane calcium entry to extrusion revealed by novel L-type calcium channel block. Cell Calcium. 44: 353-62. PMID 19230140 DOI: 10.1016/J.Ceca.2008.01.004 |
0.503 |
|
| 2008 |
Satin J, Itzhaki I, Rapoport S, Schroder EA, Izu L, Arbel G, Beyar R, Balke CW, Schiller J, Gepstein L. Calcium handling in human embryonic stem cell-derived cardiomyocytes. Stem Cells (Dayton, Ohio). 26: 1961-72. PMID 18483424 DOI: 10.1634/Stemcells.2007-0591 |
0.36 |
|
| 2008 |
Correll RN, Botzet GJ, Satin J, Andres DA, Finlin BS. Analysis of the Rem2 - voltage dependant calcium channel beta subunit interaction and Rem2 interaction with phosphorylated phosphatidylinositide lipids. Cellular Signalling. 20: 400-8. PMID 18068949 DOI: 10.1016/J.Cellsig.2007.10.029 |
0.417 |
|
| 2008 |
Correll RN, Pang C, Niedowicz DM, Satin J, Andres DA. Calmodulin binding is dispensable for Rem-mediated Ca2+ channel inhibition. Molecular and Cellular Biochemistry. 310: 103-10. PMID 18057997 DOI: 10.1007/S11010-007-9670-8 |
0.527 |
|
| 2007 |
Correll RN, Pang C, Finlin BS, Dailey AM, Satin J, Andres DA. Plasma membrane targeting is essential for Rem-mediated Ca2+ channel inhibition. The Journal of Biological Chemistry. 282: 28431-40. PMID 17686775 DOI: 10.1074/Jbc.M706176200 |
0.471 |
|
| 2007 |
Schroder E, Magyar J, Burgess D, Andres D, Satin J. Chronic verapamil treatment remodels ICa,L in mouse ventricle. American Journal of Physiology. Heart and Circulatory Physiology. 292: H1906-16. PMID 17158651 DOI: 10.1152/Ajpheart.00793.2006 |
0.521 |
|
| 2006 |
Itzhaki I, Schiller J, Beyar R, Satin J, Gepstein L. Calcium handling in embryonic stem cell-derived cardiac myocytes: of mice and men. Annals of the New York Academy of Sciences. 1080: 207-15. PMID 17132785 DOI: 10.1196/Annals.1380.017 |
0.553 |
|
| 2006 |
Schroder EA, Wei Y, Satin J. The developing cardiac myocyte: maturation of excitability and excitation-contraction coupling. Annals of the New York Academy of Sciences. 1080: 63-75. PMID 17132775 DOI: 10.1196/Annals.1380.006 |
0.493 |
|
| 2006 |
Finlin BS, Correll RN, Pang C, Crump SM, Satin J, Andres DA. Analysis of the complex between Ca2+ channel beta-subunit and the Rem GTPase. The Journal of Biological Chemistry. 281: 23557-66. PMID 16790445 DOI: 10.1074/Jbc.M604867200 |
0.47 |
|
| 2006 |
Andres DA, Crump SM, Correll RN, Satin J, Finlin BS. Analyses of Rem/RGK signaling and biological activity. Methods in Enzymology. 407: 484-98. PMID 16757347 DOI: 10.1016/S0076-6879(05)07039-4 |
0.495 |
|
| 2006 |
Crump SM, Correll RN, Schroder EA, Lester WC, Finlin BS, Andres DA, Satin J. L-type calcium channel alpha-subunit and protein kinase inhibitors modulate Rem-mediated regulation of current. American Journal of Physiology. Heart and Circulatory Physiology. 291: H1959-71. PMID 16648185 DOI: 10.1152/Ajpheart.00956.2005 |
0.551 |
|
| 2005 |
Finlin BS, Mosley AL, Crump SM, Correll RN, Ozcan S, Satin J, Andres DA. Regulation of L-type Ca2+ channel activity and insulin secretion by the Rem2 GTPase. The Journal of Biological Chemistry. 280: 41864-71. PMID 15728182 DOI: 10.1074/Jbc.M414261200 |
0.549 |
|
| 2004 |
Magyar J, Kiper CE, Dumaine R, Burgess DE, Bányász T, Satin J. Divergent action potential morphologies reveal nonequilibrium properties of human cardiac Na channels. Cardiovascular Research. 64: 477-87. PMID 15537501 DOI: 10.1016/J.Cardiores.2004.07.014 |
0.486 |
|
| 2004 |
Satin J, Kehat I, Caspi O, Huber I, Arbel G, Itzhaki I, Magyar J, Schroder EA, Perlman I, Gepstein L. Mechanism of spontaneous excitability in human embryonic stem cell derived cardiomyocytes. The Journal of Physiology. 559: 479-96. PMID 15243138 DOI: 10.1113/Jphysiol.2004.068213 |
0.433 |
|
| 2003 |
Finlin BS, Crump SM, Satin J, Andres DA. Regulation of voltage-gated calcium channel activity by the Rem and Rad GTPases. Proceedings of the National Academy of Sciences of the United States of America. 100: 14469-74. PMID 14623965 DOI: 10.1073/Pnas.2437756100 |
0.562 |
|
| 2003 |
Delisle BP, Satin J. Monovalent cations contribute to T-type calcium channel (Cav3.1 and Cav3.2) selectivity. The Journal of Membrane Biology. 193: 185-94. PMID 12962279 DOI: 10.1007/S00232-003-2017-9 |
0.699 |
|
| 2002 |
Burgess DE, Crawford O, Delisle BP, Satin J. Mechanism of inactivation gating of human T-type (low-voltage activated) calcium channels. Biophysical Journal. 82: 1894-906. PMID 11916848 DOI: 10.1016/S0006-3495(02)75539-2 |
0.691 |
|
| 2001 |
Cribbs LL, Martin BL, Schroder EA, Keller BB, Delisle BP, Satin J. Identification of the t-type calcium channel (Ca(v)3.1d) in developing mouse heart. Circulation Research. 88: 403-7. PMID 11230107 DOI: 10.1161/01.Res.88.4.403 |
0.722 |
|
| 2000 |
Satin J, Cribbs LL. Identification of a T-Type Ca2+ Channel Isoform in Murine Atrial Myocytes (AT-1 Cells) Circulation Research. 86: 636-642. PMID 10746998 DOI: 10.1161/01.Res.86.6.636 |
0.526 |
|
| 2000 |
Delisle BP, Satin J. pH modification of human T-type calcium channel gating. Biophysical Journal. 78: 1895-905. PMID 10733969 DOI: 10.1016/S0006-3495(00)76738-5 |
0.615 |
|
| 2000 |
Zhang Y, Cribbs LL, Satin J. Arachidonic acid modulation of α1H, a cloned human T-type calcium channel American Journal of Physiology-Heart and Circulatory Physiology. 278. PMID 10644598 DOI: 10.1152/Ajpheart.2000.278.1.H184 |
0.469 |
|
| 1999 |
Zhang Y, Hartmann HA, Satin J. Glycosylation influences voltage-dependent gating of cardiac and skeletal muscle sodium channels. The Journal of Membrane Biology. 171: 195-207. PMID 10501828 DOI: 10.1007/S002329900571 |
0.504 |
|
| 1998 |
Cribbs LL, Lee JH, Yang J, Satin J, Zhang Y, Daud A, Barclay J, Williamson MP, Fox M, Rees M, Perez-Reyes E. Cloning and characterization of alpha1H from human heart, a member of the T-type Ca2+ channel gene family. Circulation Research. 83: 103-9. PMID 9670923 DOI: 10.1161/01.Res.83.1.103 |
0.578 |
|
| 1996 |
Chang SY, Satin J, Fozzard HA. Modal behavior of the mu 1 Na+ channel and effects of coexpression of the beta 1-subunit. Biophysical Journal. 70: 2581-2592. PMID 8744297 DOI: 10.1016/S0006-3495(96)79829-6 |
0.374 |
|
| 1994 |
Satin J, Kyle JW, Fan Z, Rogart R, Fozzard HA, Makielski JC. Post-repolarization block of cloned sodium channels by saxitoxin: the contribution of pore-region amino acids Biophysical Journal. 66: 1353-1363. PMID 8061191 DOI: 10.1016/S0006-3495(94)80926-9 |
0.45 |
|
| 1994 |
Satin J, Limberis JT, Kyle JW, Rogart RB, Fozzard HA. The saxitoxin/tetrodotoxin binding site on cloned rat brain IIa Na channels is in the transmembrane electric field Biophysical Journal. 67: 1007-1014. PMID 7811911 DOI: 10.1016/S0006-3495(94)80566-1 |
0.449 |
|
| 1993 |
Doyle DD, Guo Y, Lustig SL, Satin J, Rogart RB, Fozzard HA. Divalent cation competition with [3H]saxitoxin binding to tetrodotoxin-resistant and -sensitive sodium channels. A two-site structural model of ion/toxin interaction. The Journal of General Physiology. 101: 153-182. PMID 8384241 DOI: 10.1085/Jgp.101.2.153 |
0.458 |
|
| 1993 |
Makielski JC, Satin J, Fan Z. Post-repolarization block of cardiac sodium channels by saxitoxin Biophysical Journal. 65: 790-798. PMID 8218903 DOI: 10.1016/S0006-3495(93)81102-0 |
0.329 |
|
| 1992 |
Satin J, Kyle JW, Chen M, Bell P, Cribbs LL, Fozzard HA, Rogart RB. A mutant of TTX-resistant cardiac sodium channels with TTX-sensitive properties. Science. 256: 1202-1205. PMID 1375397 DOI: 10.1126/Science.256.5060.1202 |
0.453 |
|
| 1992 |
Satin J, Kyle JW, Chen M, Rogart RB, Fozzard HA. The cloned cardiac Na channel α-subunit expressed in Xenopus oocytes show gating and blocking properties of native channels The Journal of Membrane Biology. 130: 11-22. PMID 1335083 DOI: 10.1007/Bf00233735 |
0.471 |
|
| 1992 |
Fozzard HA, Satin J, Chen M, Kyle J, Rogart R. Gating of the cardiac Na+ channel and drug interactions Journal of Molecular and Cellular Cardiology. 24: 23. DOI: 10.1016/0022-2828(92)90099-L |
0.443 |
|
| 1990 |
Cribbs LL, Satin J, Fozzard HA, Rogart RB. Functional expression of the rat heart I Na+ channel isoform. Demonstration of properties characteristic of native cardiac Na+ channels. Febs Letters. 275: 195-200. PMID 2175715 DOI: 10.1016/0014-5793(90)81470-9 |
0.427 |
|
| 1990 |
DeHaan RL, Fujii S, Satin J. Cell Interactions in Cardiac Development Development Growth & Differentiation. 32: 233-241. DOI: 10.1111/J.1440-169X.1990.00233.X |
0.448 |
|
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