Steven D. Cappell, Ph.D.
Affiliations: | 2010 | Pharmacology | University of North Carolina, Chapel Hill, Chapel Hill, NC |
Area:
Pharmacology, BiochemistryGoogle:
"Steven Cappell"Mean distance: (not calculated yet)
Parents
Sign in to add mentor| Henrik G. Dohlman | grad student | 2010 | UNC Chapel Hill | |
| (Systematic analysis of essential genes reveals new regulators of G protein signaling.) | ||||
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Publications
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| Cornwell JA, Crncec A, Afifi MM, et al. (2024) Author Correction: Loss of CDK4/6 activity in S/G2 phase leads to cell cycle reversal. Nature |
| Lang F, Cornwell JA, Kaur K, et al. (2023) Abrogation of the G2/M checkpoint as a chemo sensitization approach for alkylating agents. Neuro-Oncology |
| Cornwell JA, Crncec A, Afifi MM, et al. (2023) Loss of CDK4/6 activity in S/G2 phase leads to cell cycle reversal. Nature |
| Awadia S, Sitto M, Ram S, et al. (2023) The adapter protein FADD provides an alternate pathway for entry into the cell cycle by regulating APC/C-Cdh1 E3 ubiquitin ligase activity. The Journal of Biological Chemistry. 104786 |
| Nathans JF, Cornwell JA, Afifi MM, et al. (2021) Cell cycle inertia underlies a bifurcation in cell fates after DNA damage. Science Advances. 7 |
| Yang HW, Cappell SD, Jaimovich A, et al. (2020) Stress-mediated exit to quiescence restricted by increasing persistence in CDK4/6 activation. Elife. 9 |
| Chung M, Liu C, Yang HW, et al. (2019) Transient Hysteresis in CDK4/6 Activity Underlies Passage of the Restriction Point in G1. Molecular Cell |
| Cappell SD, Mark KG, Garbett D, et al. (2018) EMI1 switches from being a substrate to an inhibitor of APC/C to start the cell cycle. Nature |
| Cappell SD, Chung M, Jaimovich A, et al. (2016) Irreversible APC(Cdh1) Inactivation Underlies the Point of No Return for Cell-Cycle Entry. Cell. 166: 167-180 |
| Spencer SL, Cappell SD, Tsai FC, et al. (2013) The proliferation-quiescence decision is controlled by a bifurcation in CDK2 activity at mitotic exit. Cell. 155: 369-83 |