Amanda R. Wasylishen, Ph.D.

Affiliations: 
2012 University of Toronto, Toronto, ON, Canada 
Area:
General Biophysics
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Parents

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Linda Z. Penn grad student 2012 University of Toronto
 (Characterizing the Mechanisms Regulating Myc-Induced Transformation.)
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Publications

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Moyer SM, Wasylishen AR, Qi Y, et al. (2020) p53 drives a transcriptional program that elicits a non-cell-autonomous response and alters cell state in vivo. Proceedings of the National Academy of Sciences of the United States of America
Wasylishen AR, Sun C, Moyer SM, et al. (2020) Daxx maintains endogenous retroviral silencing and restricts cellular plasticity in vivo. Science Advances. 6: eaba8415
Wasylishen AR, Sun C, Chau GP, et al. (2020) Men1 maintains exocrine pancreas homeostasis in response to inflammation and oncogenic stress. Proceedings of the National Academy of Sciences of the United States of America
El Beaino M, Liu J, Wasylishen AR, et al. (2020) Loss of Stag2 cooperates with EWS-FLI1 to transform murine Mesenchymal stem cells. Bmc Cancer. 20: 3
Pant V, Xiong S, Wasylishen AR, et al. (2019) Transient enhancement of p53 activity protects from radiation-induced gastrointestinal toxicity. Proceedings of the National Academy of Sciences of the United States of America
Samdani RT, Wasylishen AR, Halperin DM, et al. (2019) Loss of Menin Expression by Immunohistochemistry in Pancreatic Neuroendocrine Tumors: Comparison Between Primary and Metastatic Tumors. Pancreas
Aryal NK, Pant V, Wasylishen AR, et al. (2019) Dicer1 phospho-mimetic promotes tumor progression and dissemination. Cancer Research
Ding J, Li X, Zhang C, et al. (2019) CBMT-32. EGFR SUPPRESSES p53 FUNCTION THROUGH DNA-PKcs BINDING TO p53: NOVEL CROSSTALK BETWEEN EGFR AND TP53 IN GLIOBLASTOMA Neuro-Oncology. 21: vi40-vi40
Aryal NK, Pant V, Wasylishen AR, et al. (2018) Constitutive Dicer1 phosphorylation accelerates metabolism and aging in vivo. Proceedings of the National Academy of Sciences of the United States of America
Wasylishen AR, Estrella JS, Pant V, et al. (2018) Daxx Functions are p53-independent In Vivo. Molecular Cancer Research : McR
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