Judd M. Aiken

University of Wisconsin, Madison, Madison, WI 
Cell Biology, Molecular Biology, General Biophysics
"Judd Aiken"
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Volga A. Iniguez Rojas grad student 2000 UW Madison
Marisol E. Lopez grad student 2000 UW Madison
Jonathan Wanagat grad student 2000 UW Madison
Entela A. Bua grad student 2004 UW Madison
Allen J. Herbst grad student 2008 UW Madison
Laura R. Moody grad student 2010 UW Madison
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Kuznetsova A, Cullingham C, McKenzie D, et al. (2018) Soil humic acids degrade CWD prions and reduce infectivity. Plos Pathogens. 14: e1007414
Gushue D, Herbst A, Sim V, et al. (2018) 14-3-3 and Enolase Abundances in the CSF of Prion Diseased Rats. Prion
Bielas J, Herbst A, Widjaja K, et al. (2018) Long term rapamycin treatment improves mitochondrial DNA quality in aging mice. Experimental Gerontology
Wei L, Gregorich ZR, Lin Z, et al. (2017) Novel Sarcopenia-related Alterations in Sarcomeric Protein Post-translational Modifications in Skeletal Muscles Identified by Top-down Proteomics. Molecular & Cellular Proteomics : McP
Herbst A, Velásquez CD, Triscott E, et al. (2017) Chronic Wasting Disease Prion Strain Emergence and Host Range Expansion. Emerging Infectious Diseases. 23: 1598-1600
Herbst A, Widjaja K, Nguy B, et al. (2017) Digital PCR Quantitation of Muscle Mitochondrial DNA: Age, Fiber Type, and Mutation-Induced Changes. The Journals of Gerontology. Series a, Biological Sciences and Medical Sciences
Barton GP, Sepe JJ, McKiernan SH, et al. (2016) Mitochondrial and Metabolic Gene Expression in the Aged Rat Heart. Frontiers in Physiology. 7: 352
Herbst A, Wanagat J, Cheema N, et al. (2016) Latent mitochondrial DNA deletion mutations drive muscle fiber loss at old age. Aging Cell
Gregorich ZR, Peng Y, Cai W, et al. (2016) Top-down Targeted Proteomics Reveals Decrease in Myosin Regulatory Light Chain Phosphorylation that Contributes to Sarcopenic Muscle Dysfunction. Journal of Proteome Research
Cheema N, Herbst A, McKenzie D, et al. (2015) Apoptosis and necrosis mediate skeletal muscle fiber loss in age-induced mitochondrial enzymatic abnormalities. Aging Cell
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