Jabe M. Best, Ph.D.
|2011||University of Wisconsin, Madison, Madison, WI|
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|Tyan L, Foell JD, Vincent KP, et al. (2018) Long QT syndrome caveolin-3 mutations differentially modulate K 4 and Ca 1.2 channels to contribute to action potential prolongation. The Journal of Physiology|
|Boczek NJ, Best JM, Tester DJ, et al. (2013) Exome sequencing and systems biology converge to identify novel mutations in the L-type calcium channel, CACNA1C, linked to autosomal dominant long QT syndrome. Circulation. Cardiovascular Genetics. 6: 279-89|
|Best JM, Kamp TJ. (2012) Different subcellular populations of L-type Ca2+ channels exhibit unique regulation and functional roles in cardiomyocytes. Journal of Molecular and Cellular Cardiology. 52: 376-87|
|Best JM, Foell JD, Buss CR, et al. (2011) Small GTPase Rab11b regulates degradation of surface membrane L-type Cav1.2 channels. American Journal of Physiology. Cell Physiology. 300: C1023-33|
|Best JM, Kamp TJ. (2010) A sympathetic model of L-type Ca2+ channel-triggered arrhythmias. American Journal of Physiology. Heart and Circulatory Physiology. 298: H3-4|
|Delisle BP, Underkofler HA, Moungey BM, et al. (2009) Small GTPase determinants for the Golgi processing and plasmalemmal expression of human ether-a-go-go related (hERG) K+ channels. The Journal of Biological Chemistry. 284: 2844-53|