Sarah Ann Boswell
Affiliations: | 2006 | Medical Physics | University of Wisconsin, Madison, Madison, WI |
Website:
https://www.linkedin.com/in/sarah-boswell-070139115Google:
"Sarah Ann Boswell"Bio:
https://www.medphysics.wisc.edu/grads/abstracts/Boswell_Sarah_2006_abstract_KDW.pdf
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Parents
Sign in to add mentor| Thomas Rockwell Mackie | grad student | 2006 | UW Madison | |
| (Improving geometric accuracy in radiation delivery using helical tomotherapy.) | ||||
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Publications
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| Schauer NJ, Liu X, Magin RS, et al. (2020) Selective USP7 inhibition elicits cancer cell killing through a p53-dependent mechanism. Scientific Reports. 10: 5324 |
| Hafner M, Mills CE, Subramanian K, et al. (2019) Multiomics Profiling Establishes the Polypharmacology of FDA-Approved CDK4/6 Inhibitors and the Potential for Differential Clinical Activity. Cell Chemical Biology |
| Wang H, Sheehan RP, Palmer AC, et al. (2019) Adaptation of Human iPSC-Derived Cardiomyocytes to Tyrosine Kinase Inhibitors Reduces Acute Cardiotoxicity via Metabolic Reprogramming. Cell Systems |
| Mills CE, Hafner MA, Subramanian K, et al. (2019) Abstract 4432: Multi-omics profiling establishes the polypharmacology of FDA Approved CDK4/6 inhibitors and its impact on drug response Cancer Research. 79: 4432-4432 |
| Mills C, Hafner M, Subramanian K, et al. (2018) Abstract PD1-12: Omics profiling of CDK4/6 inhibitors reveals functionally important secondary targets of abemaciclib Cancer Research. 16 |
| Hafner M, Mills C, Subramanian K, et al. (2018) Abstract PD4-02: Advantageous polypharmacology of abemaciclib revealed by omics profiling of CDK4/6 inhibitors Cancer Research. 78 |
| Fallahi-Sichani M, Becker V, Izar B, et al. (2017) Adaptive resistance of melanoma cells to RAF inhibition via reversible induction of a slowly dividing de-differentiated state. Molecular Systems Biology. 13: 905 |
| Fallahi-Sichani M, Becker V, Izar B, et al. (2017) Abstract PR17: Single-cell analysis reveals an adaptive, slowly-dividing, de-differentiated, drug-resistant cell state selectively inhibitable by drug combinations Molecular Cancer Therapeutics. 16 |
| Brown L, Boswell S, Raj L, et al. (2007) Transcriptional targets of p53 that regulate cellular proliferation Critical Reviews in Eukaryotic Gene Expression. 17: 73-85 |
| Boswell SA, Ongusaha PP, Nghiem P, et al. (2007) The protective role of a small GTPase RhoE against UVB-induced DNA damage in keratinocytes. The Journal of Biological Chemistry. 282: 4850-8 |