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Rebecca L. Hutcheson, Ph.D. - Publications

Affiliations: 
2013 College of Medicine University of South Alabama, Mobile, AL, United States 
Area:
Biochemistry, Medicine and Surgery, Molecular Biology
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9 high-probability publications. We are testing a new system for linking publications to authors. You can help! If you notice any inaccuracies, please sign in and mark papers as correct or incorrect matches. If you identify any major omissions or other inaccuracies in the publication list, please let us know.

Year Citation  Score
2018 Soler A, Hunter I, Joseph G, Hutcheson R, Hutcheson B, Yang J, Zhang FF, Joshi SR, Bradford C, Gotlinger KH, Maniyar R, Falck JR, Proctor S, Schwartzman ML, Gupte SA, et al. Corrigendum to "Elevated 20-HETE in metabolic syndrome regulates arterial stiffness and systolic hypertension via MMP12 activation" [J. Mol. Cell. Cardiol. 117 (2018) 88-99]. Journal of Molecular and Cellular Cardiology. PMID 29625729 DOI: 10.1016/J.Yjmcc.2018.03.015  0.731
2018 Soler A, Hunter I, Joseph G, Hutcheson R, Hutcheson B, Yang J, Zhang FF, Joshi SR, Bradford C, Gotlinger KH, Maniyar R, Falck JR, Proctor S, Schwartzman ML, Gupte SA, et al. Elevated 20-HETE in metabolic syndrome regulates arterial stiffness and systolic hypertension via MMP12 activation. Journal of Molecular and Cellular Cardiology. PMID 29428638 DOI: 10.1016/J.Yjmcc.2018.02.005  0.732
2016 Joseph G, Soler A, Hutcheson R, Hunter I, Bradford C, Hutcheson B, Gotlinger KH, Jiang H, Falck JR, Proctor S, Laniado Schwartzman M, Rocic P. Elevated 20-HETE Impairs Coronary Collateral Growth in Metabolic Syndrome Via Endothelial Dysfunction. American Journal of Physiology. Heart and Circulatory Physiology. ajpheart.00561.2016. PMID 28011587 DOI: 10.1152/Ajpheart.00561.2016  0.725
2016 Diane A, Pierce WD, Kelly SE, Sokolik S, Borthwick F, Jacome-Sosa M, Mangat R, Pradillo JM, Allan SM, Ruth MR, Field CJ, Hutcheson R, Rocic P, Russell JC, Vine DF, et al. Mechanisms of Comorbidities Associated With the Metabolic Syndrome: Insights from the JCR:LA-cp Corpulent Rat Strain. Frontiers in Nutrition. 3: 44. PMID 27777929 DOI: 10.3389/Fnut.2016.00044  0.691
2015 Hutcheson R, Terry R, Hutcheson B, Jadhav R, Chaplin J, Smith E, Barrington R, Proctor SD, Rocic P. miR-21-mediated decreased neutrophil apoptosis is a determinant of impaired coronary collateral growth in metabolic syndrome. American Journal of Physiology. Heart and Circulatory Physiology. 308: H1323-35. PMID 25840830 DOI: 10.1152/Ajpheart.00654.2014  0.717
2014 Hutcheson R, Chaplin J, Hutcheson B, Borthwick F, Proctor S, Gebb S, Jadhav R, Smith E, Russell JC, Rocic P. miR-21 normalizes vascular smooth muscle proliferation and improves coronary collateral growth in metabolic syndrome. Faseb Journal : Official Publication of the Federation of American Societies For Experimental Biology. 28: 4088-99. PMID 24903275 DOI: 10.1096/Fj.14-251223  0.76
2013 Dodd T, Wiggins L, Hutcheson R, Smith E, Musiyenko A, Hysell B, Russell JC, Rocic P. Impaired coronary collateral growth in the metabolic syndrome is in part mediated by matrix metalloproteinase 12-dependent production of endostatin and angiostatin. Arteriosclerosis, Thrombosis, and Vascular Biology. 33: 1339-49. PMID 23599440 DOI: 10.1161/Atvbaha.113.301533  0.611
2013 Hutcheson R, Terry R, Chaplin J, Smith E, Musiyenko A, Russell JC, Lincoln T, Rocic P. MicroRNA-145 restores contractile vascular smooth muscle phenotype and coronary collateral growth in the metabolic syndrome. Arteriosclerosis, Thrombosis, and Vascular Biology. 33: 727-36. PMID 23393394 DOI: 10.1161/Atvbaha.112.301116  0.721
2012 Hutcheson R, Rocic P. The metabolic syndrome, oxidative stress, environment, and cardiovascular disease: the great exploration. Experimental Diabetes Research. 2012: 271028. PMID 22829804 DOI: 10.1155/2012/271028  0.708
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