Neelkantha Sen, PhD - Publications

Affiliations: 
2012- Department of Neuroscience & Degenerative Medicine Augusta University 

15 high-probability publications. We are testing a new system for linking publications to authors. You can help! If you notice any inaccuracies, please sign in and mark papers as correct or incorrect matches. If you identify any major omissions or other inaccuracies in the publication list, please let us know.

Year Citation  Score
2021 Sen T, Sen N. Corrigendum to 'Treatment with an activator of hypoxia-inducible factor 1, DMOG provides neuroprotection after traumatic brain injury' [Neuropharmacology 107 (2016) 79-88]. Neuropharmacology. 108536. PMID 34247851 DOI: 10.1016/j.neuropharm.2021.108536  0.419
2019 Sen T, Saha P, Gupta R, Foley LM, Jiang T, Abakumova OS, Hitchens KT, Sen N. Aberrant ER-stress induced neuronal-IFNβ elicits white matter injury due to microglial activation and T-cell infiltration after TBI. The Journal of Neuroscience : the Official Journal of the Society For Neuroscience. PMID 31694961 DOI: 10.1523/JNEUROSCI.0718-19.2019  0.708
2019 Saha P, Gupta RK, Sen T, Sen N. Histone deacetylase 4 downregulation elicits post-traumatic psychiatric disorders through impairment of neurogenesis. Journal of Neurotrauma. PMID 31169064 DOI: 10.1089/neu.2019.6373  0.705
2019 Gupta R, Saha P, Sen T, Sen N. Corrigendum to "An augmentation in histone dimethylation at lysine nine residues elicits vision impairment following traumatic brain injury" [Free Radic. Biol. Med. (2019 Feb 18) 134 630-643]. Free Radical Biology & Medicine. PMID 31003850 DOI: 10.1016/j.freeradbiomed.2019.04.010  0.396
2019 Sen N. Aberrant cell cycle induction is pivotal for mitochondrial biogenesis after traumatic brain injury. Neural Regeneration Research. 14: 1215-1216. PMID 30804250 DOI: 10.4103/1673-5374.251305  0.397
2019 Gupta R, Saha P, Sen T, Sen N. An augmentation in histone dimethylation at lysine nine residues elicits vision impairment following traumatic brain injury. Free Radical Biology & Medicine. PMID 30790655 DOI: 10.1016/j.freeradbiomed.2019.02.015  0.669
2018 Sen N. ER Stress, CREB, and Memory: A Tangled Emerging Link in Disease. The Neuroscientist : a Review Journal Bringing Neurobiology, Neurology and Psychiatry. 1073858418816611. PMID 30477403 DOI: 10.1177/1073858418816611  0.311
2018 Saha P, Gupta R, Sen T, Sen N. Activation of cyclin D1 affects mitochondrial mass following traumatic brain injury. Neurobiology of Disease. PMID 30010002 DOI: 10.1016/j.nbd.2018.07.010  0.693
2017 Sen T, Gupta R, Kaiser H, Sen N. Activation of PERK elicits memory impairment through inactivation of CREB and downregulation of PSD95 following Traumatic Brain Injury. The Journal of Neuroscience : the Official Journal of the Society For Neuroscience. PMID 28522733 DOI: 10.1523/JNEUROSCI.2343-16.2017  0.698
2017 Sen N. An insight into the vision impairment following traumatic brain injury. Neurochemistry International. PMID 28163060 DOI: 10.1016/j.neuint.2017.01.019  0.413
2016 Sen T, Sen N. Isoflurane-induced inactivation of CREB through histone deacetylase 4 is responsible for cognitive impairment in developing brain. Neurobiology of Disease. PMID 27544482 DOI: 10.1016/j.nbd.2016.08.005  0.363
2016 Sen T, Sen N. Treatment with an activator of hypoxia-inducible factor 1, DMOG provides neuroprotection after traumatic brain injury. Neuropharmacology. PMID 26970014 DOI: 10.1016/j.neuropharm.2016.03.009  0.424
2015 Gupta R, Sen N. Traumatic brain injury: a risk factor for neurodegenerative diseases. Reviews in the Neurosciences. PMID 26352199 DOI: 10.1515/revneuro-2015-0017  0.69
2013 Kapoor S, Kim SM, Farook JM, Mir S, Saha R, Sen N. Foxo3a transcriptionally upregulates AQP4 and induces cerebral edema following traumatic brain injury. The Journal of Neuroscience : the Official Journal of the Society For Neuroscience. 33: 17398-403. PMID 24174672 DOI: 10.1523/JNEUROSCI.2756-13.2013  0.461
2013 Farook JM, Shields J, Tawfik A, Markand S, Sen T, Smith SB, Brann D, Dhandapani KM, Sen N. Erratum: GADD34 induces cell death through inactivation of Akt following traumatic brain injury Cell Death & Disease. 4: e813-e813. DOI: 10.1038/Cddis.2013.336  0.425
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