Eric J. Gapud, Ph.D.

Affiliations: 
2011 Biology & Biomedical Sciences (Immunology) Washington University, Saint Louis, St. Louis, MO 
Area:
Immunology, Molecular Biology, Genetics
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"Eric Gapud"

Parents

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Barry Sleckman grad student 2011 Washington University
 (Regulation of DNA repair by Atm and DNA-PKcs during V(D)J recombination.)
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Publications

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Gapud EJ, Trejo-Zambrano MI, Gomez-Banuelos E, et al. (2020) Granzyme B Induces IRF-3 Phosphorylation through a Perforin-Independent Proteolysis-Dependent Signaling Cascade without Inducing Cell Death. Journal of Immunology (Baltimore, Md. : 1950)
Lee BS, Gapud EJ, Zhang S, et al. (2013) Functional intersection of ATM and DNA-dependent protein kinase catalytic subunit in coding end joining during V(D)J recombination. Molecular and Cellular Biology. 33: 3568-79
Gapud EJ, Lee BS, Mahowald GK, et al. (2011) Repair of chromosomal RAG-mediated DNA breaks by mutant RAG proteins lacking phosphatidylinositol 3-like kinase consensus phosphorylation sites. Journal of Immunology (Baltimore, Md. : 1950). 187: 1826-34
Gapud EJ, Sleckman BP. (2011) Unique and redundant functions of ATM and DNA-PKcs during V(D)J recombination. Cell Cycle (Georgetown, Tex.). 10: 1928-35
Zhang S, Yajima H, Huynh H, et al. (2011) Congenital bone marrow failure in DNA-PKcs mutant mice associated with deficiencies in DNA repair. The Journal of Cell Biology. 193: 295-305
Gapud EJ, Dorsett Y, Yin B, et al. (2011) Ataxia telangiectasia mutated (Atm) and DNA-PKcs kinases have overlapping activities during chromosomal signal joint formation. Proceedings of the National Academy of Sciences of the United States of America. 108: 2022-7
Gonzalez-Suarez I, Redwood AB, Perkins SM, et al. (2009) Novel roles for A-type lamins in telomere biology and the DNA damage response pathway. The Embo Journal. 28: 2414-27
Difilippantonio S, Gapud E, Wong N, et al. (2008) 53BP1 facilitates long-range DNA end-joining during V(D)J recombination. Nature. 456: 529-33
Bredemeyer AL, Helmink BA, Innes CL, et al. (2008) DNA double-strand breaks activate a multi-functional genetic program in developing lymphocytes. Nature. 456: 819-23
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