Simon M. Harrison

University of Leeds, Leeds, England, United Kingdom 
"Simon Harrison"
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Duncan DJ, Yang Z, Hopkins PM, et al. (2010) TNF-alpha and IL-1beta increase Ca2+ leak from the sarcoplasmic reticulum and susceptibility to arrhythmia in rat ventricular myocytes. Cell Calcium. 47: 378-86
Zhang H, Tao T, Kharche S, et al. (2009) Modelling changes in transmural propagation and susceptibility to arrhythmia induced by volatile anaesthetics in ventricular tissue. Journal of Theoretical Biology. 257: 279-91
Stones R, Calaghan SC, Billeter R, et al. (2007) Transmural variations in gene expression of stretch-modulated proteins in the rat left ventricle. PflüGers Archiv : European Journal of Physiology. 454: 545-9
Duncan DJ, Hopkins PM, Harrison SM. (2007) Negative inotropic effects of tumour necrosis factor-alpha and interleukin-1beta are ameliorated by alfentanil in rat ventricular myocytes. British Journal of Pharmacology. 150: 720-6
Fowler MR, Naz JR, Graham MD, et al. (2007) Age and hypertrophy alter the contribution of sarcoplasmic reticulum and Na+/Ca2+ exchange to Ca2+ removal in rat left ventricular myocytes. Journal of Molecular and Cellular Cardiology. 42: 582-9
Fowler MR, Orchard CH, Harrison SM. (2007) Cellular distribution of calcium current is unaltered during compensated hypertrophy in the spontaneously hypertensive rat. PflüGers Archiv : European Journal of Physiology. 453: 463-9
Graham MD, Hopkins PM, Harrison SM. (2006) Antagonistic actions of halothane and sevoflurane on spontaneous Ca2+ release in rat ventricular myocytes. Anesthesiology. 105: 58-64
Bru-Mercier G, Hopkins PM, Harrison SM. (2005) Halothane and sevoflurane inhibit Na/Ca exchange current in rat ventricular myocytes. British Journal of Anaesthesia. 95: 305-9
Yang Z, Harrison SM, Steele DS. (2005) ATP-dependent effects of halothane on SR Ca2+ regulation in permeabilized atrial myocytes. Cardiovascular Research. 65: 167-76
Fowler MR, Naz JR, Graham MD, et al. (2005) Decreased Ca2+ extrusion via Na+/Ca2+ exchange in epicardial left ventricular myocytes during compensated hypertrophy. American Journal of Physiology. Heart and Circulatory Physiology. 288: H2431-8
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