Steven Marston

Affiliations: 
National Heart & Lung Institute Imperial College London, London, England, United Kingdom 
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"Steven Marston"

Parents

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Richard T. Tregear grad student Imperial College London
Edwin W. Taylor post-doc 1976- Chicago (Cell Biology Tree)
Annemarie Weber post-doc 1974-1975 Penn (Neurotree)
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Publications

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Piroddi N, Witjas-Paalberends ER, Ferrara C, et al. (2018) The homozygous K280N troponin T mutation alters cross-bridge kinetics and energetics in human HCM. The Journal of General Physiology
Rynkiewicz MJ, Prum T, Hollenberg S, et al. (2017) Tropomyosin Must Interact Weakly with Actin to Effectively Regulate Thin Filament Function. Biophysical Journal. 113: 2444-2451
Toepfer CN, Sikkel MB, Caorsi V, et al. (2016) A post-MI power struggle: adaptations in cardiac power occur at the sarcomere level alongside MyBP-C and RLC phosphorylation. American Journal of Physiology. Heart and Circulatory Physiology. ajpheart.00899.2015
Chan C, Fan J, Messer AE, et al. (2016) Myopathy-inducing mutation H40Y in ACTA1 hampers actin filament structure and function. Biochimica Et Biophysica Acta
Messer AE, Bayliss CR, El-Mezgueldi M, et al. (2016) Mutations in troponin T associated with Hypertrophic Cardiomyopathy increase Ca(2+)-sensitivity and suppress the modulation of Ca(2+)-sensitivity by troponin I phosphorylation. Archives of Biochemistry and Biophysics
Marston S, Messer A, Papadaki M. (2015) (De-)sensitization vs. uncoupling: what drives cardiomyopathies in the thin filament? Reply. Cardiovascular Research
Wilkinson R, Song W, Smoktunowicz N, et al. (2015) A DILATED CARDIOMYOPATHY MUTATION BLUNTS ADRENERGIC RESPONSE AND INDUCES CONTRACTILE DYSFUNCTION UNDER CHRONIC ANGIOTENSIN II STRESS. American Journal of Physiology. Heart and Circulatory Physiology. ajpheart.00327.2015
Donkervoort S, Papadaki M, de Winter JM, et al. (2015) TPM3 deletions cause a hypercontractile congenital muscle stiffness phenotype. Annals of Neurology
Marston S, Montgiraud C, Munster AB, et al. (2015) OBSCN Mutations Associated with Dilated Cardiomyopathy and Haploinsufficiency. Plos One. 10: e0138568
Yuen M, Cooper ST, Marston SB, et al. (2015) Muscle weakness in TPM3-myopathy is due to reduced Ca2+-sensitivity and impaired acto-myosin cross-bridge cycling in slow fibres. Human Molecular Genetics
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