Harris Joseph Granger, Ph.D

Affiliations:

1976-2018

Medical Physiology, College of Medicine

Texas A & M University, College Station, TX, United States

Area:

Cardiovascular Physiology, Cell Biology, Microcirculation

Website:

https://medicine.tamu.edu/centers/2023-CVRI-Symposium-Booklet-compressed.pdf

Google:

""Harris j Granger""

Bio:

(1944 - 2018)

Associate Professor of Medical Physiology, College of Medicine, Texas A&M University, 1976-78
Professor of Medical Physiology, College of Medicine, Texas A&M University, 1978-2018
Director, Microcirculation Research Institute, College of Medicine, Texas A&M University, 1981-1998.
Head, Department of Medical Physiology, College of Medicine, Texas A&M University, 1982-2014
Distinguished Professor, Texas A&M University Health Science Center, 1997-2018
Director, Cardiovascular Research Institute, Texas A&M University Health Science Center, 1998-2018

Dr. Harris Joseph Granger, age 73, passed away peacefully on August 21, 2018, surrounded by his family, following a lengthy battle with several health issues.
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Cross-listing: Computational Biology Tree - Cell Biology Tree

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Parents

Arthur Clifton Guyton	grad student	1966-1970	University of Mississippi Medical Center
Benjamin William Zweifach	post-doc	1975-1976	University of California at San Diego School of Medicine
(NIH Postdoctoral Fellow, Sabbatical)

Children

Glen A. Laine	grad student	1980	Texas A & M
Cynthia Meininger	post-doc		Texas A & M College of Medicine (Cell Biology Tree)
Gerald A. Meininger	post-doc		Texas A & M College of Medicine (Cell Biology Tree)
Sarah Y. Yuan	post-doc		Texas A & M (Cell Biology Tree)

Collaborators

BETA: Related publications

Publications

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Donnini S, Solito R, Giachetti A, et al. (2006) Fibroblast growth factor-2 mediates angiotensin-converting enzyme inhibitor-induced angiogenesis in coronary endothelium Journal of Pharmacology and Experimental Therapeutics. 319: 515-522
Moy AB, Blackwell K, Wu MH, et al. (2006) Growth factor- and heparin-dependent regulation of constitutive and agonist-mediated human endothelial barrier function American Journal of Physiology - Heart and Circulatory Physiology. 291
Dawson NS, Zawieja DC, Wu MH, et al. (2006) Signaling pathways mediating VEGF165-induced calcium transients and membrane depolarization in human endothelial cells. Faseb Journal : Official Publication of the Federation of American Societies For Experimental Biology. 20: 991-3
Guo M, Wu MH, Granger HJ, et al. (2005) Focal adhesion kinase in neutrophil-induced microvascular hyperpermeability. Microcirculation (New York, N.Y. : 1994). 12: 223-32
Steinle JJ, Zamora DO, Rosenbaum JT, et al. (2005) Beta 3-adrenergic receptors mediate choroidal endothelial cell invasion, proliferation, and cell elongation. Experimental Eye Research. 80: 83-91
Wu MH, Yuan SY, Granger HJ. (2005) The protein kinase MEK1/2 mediate vascular endothelial growth factor- and histamine-induced hyperpermeability in porcine coronary venules Journal of Physiology. 563: 95-104
Guo M, Wu MH, Granger HJ, et al. (2004) Transference of recombinant VE-cadherin cytoplasmic domain alters endothelial junctional integrity and porcine microvascular permeability. The Journal of Physiology. 554: 78-88
Steinle JJ, Granger HJ. (2003) Nerve growth factor regulates human choroidal, but not retinal, endothelial cell migration and proliferation. Autonomic Neuroscience : Basic & Clinical. 108: 57-62
Steinle JJ, Meininger CJ, Chowdhury U, et al. (2003) Role of ephrin B2 in human retinal endothelial cell proliferation and migration. Cellular Signalling. 15: 1011-7
Wu MH, Guo M, Yuan SY, et al. (2003) Focal adhesion kinase mediates porcine venular hyperpermeability elicited by vascular endothelial growth factor. The Journal of Physiology. 552: 691-9