| Year |
Citation |
Score |
| 2017 |
Veltri T, Landim-Vieira M, Parvatiyar MS, Gonzalez-Martinez D, Dieseldorff Jones KM, Michell CA, Dweck D, Landstrom AP, Chase PB, Pinto JR. Hypertrophic Cardiomyopathy Cardiac Troponin C Mutations Differentially Affect Slow Skeletal and Cardiac Muscle Regulation. Frontiers in Physiology. 8: 221. PMID 28473771 DOI: 10.3389/Fphys.2017.00221 |
0.758 |
|
| 2015 |
Vukmirovic M, Sanchez-Gonzalez MA, Frye GS, Koutnik A, Gonzalez DA, Ruiz E, Krivensky E, Dweck D, Ferreira LF, Pinto JR. Structural and Functional Changes in Skeletal Muscles in an A8V-Troponin C Hypertrophic Cardiomyopathy Knock-In Mouse Model Biophysical Journal. 108: 594a. DOI: 10.1016/J.Bpj.2014.11.3233 |
0.493 |
|
| 2015 |
Veltri T, Michell CA, Landim M, Dweck D, Pinto JR. The Functional Consequences of Hypertrophic Cardiomyopathy Troponin C Mutations in the Regulation of Slow Skeletal Muscle Contraction: The Protective Role of Slow Skeletal Troponin I Biophysical Journal. 108: 593a-594a. DOI: 10.1016/J.Bpj.2014.11.3232 |
0.695 |
|
| 2014 |
Dweck D, Sanchez-Gonzalez MA, Chang AN, Dulce RA, Badger CD, Koutnik AP, Ruiz EL, Griffin B, Liang J, Kabbaj M, Fincham FD, Hare JM, Overton JM, Pinto JR. Long term ablation of protein kinase A (PKA)-mediated cardiac troponin I phosphorylation leads to excitation-contraction uncoupling and diastolic dysfunction in a knock-in mouse model of hypertrophic cardiomyopathy. The Journal of Biological Chemistry. 289: 23097-111. PMID 24973218 DOI: 10.1074/Jbc.M114.561472 |
0.63 |
|
| 2014 |
Vukmirovic M, Sanchez-Gonzalez MA, Ruiz E, Krivensky E, Dweck D, Pinto JR. Myosin Heavy Chain Isoform Switching in Skeletal Muscles in an A8V-Troponin C Hypertrophic Cardiomyopathy Knock-In Mouse Model Biophysical Journal. 106: 775a. DOI: 10.1016/J.Bpj.2013.11.4253 |
0.479 |
|
| 2014 |
Martins AS, Parvatiyar MS, Turna R, Badger CD, Griffin B, Zorio D, Vukmirovic M, Sanchez-Gonzalez MA, Dweck D, Ruiz EL, Liang J, Wang Y, Overton J, Pinto JR. In Vivo Analysis of Troponin C Knock-In (A8V) Mice: Evidence that TNNC1 is a Hypertrophic Cardiomyopathy Susceptibility Gene Biophysical Journal. 106: 723a. DOI: 10.1016/J.Bpj.2013.11.3995 |
0.758 |
|
| 2014 |
Turna RS, Weller RS, Hwang H, Dweck D, Ahmad F, Huke S, Knollmann BC, Pinto JR. Crossing Mice Carrying TnT Disease Mutations with Opposite Effects on the Myofilament Calcium Sensitivity Partially Normalizes Myofilament Function and Ameliorates Cardiomyopathy Phenotypes Biophysical Journal. 106: 345a. DOI: 10.1016/J.Bpj.2013.11.1968 |
0.639 |
|
| 2012 |
Wang Y, Pinto JR, Solis RS, Dweck D, Liang J, Diaz-Perez Z, Ge Y, Walker JW, Potter JD. Generation and functional characterization of knock-in mice harboring the cardiac troponin I-R21C mutation associated with hypertrophic cardiomyopathy. The Journal of Biological Chemistry. 287: 2156-67. PMID 22086914 DOI: 10.1074/Jbc.M111.294306 |
0.651 |
|
| 2012 |
Dweck D, Liang J, Potter JD, Pinto JR. The Functional Consequences of HCM Troponin C Mutations in the Regulation of Slow Skeletal Muscle Contraction Biophysical Journal. 102: 155a. DOI: 10.1016/J.Bpj.2011.11.848 |
0.678 |
|
| 2011 |
Pinto JR, Reynaldo DP, Parvatiyar MS, Dweck D, Liang J, Jones MA, Sorenson MM, Potter JD. Strong cross-bridges potentiate the Ca(2+) affinity changes produced by hypertrophic cardiomyopathy cardiac troponin C mutants in myofilaments: a fast kinetic approach. The Journal of Biological Chemistry. 286: 1005-13. PMID 21056975 DOI: 10.1074/Jbc.M110.168583 |
0.732 |
|
| 2010 |
Parvatiyar MS, Pinto JR, Dweck D, Potter JD. Cardiac troponin mutations and restrictive cardiomyopathy. Journal of Biomedicine & Biotechnology. 2010: 350706. PMID 20617149 DOI: 10.1155/2010/350706 |
0.716 |
|
| 2010 |
Dweck D, Reynaldo DP, Pinto JR, Potter JD. A dilated cardiomyopathy troponin C mutation lowers contractile force by reducing strong myosin-actin binding. The Journal of Biological Chemistry. 285: 17371-9. PMID 20371872 DOI: 10.1074/Jbc.M109.064105 |
0.69 |
|
| 2010 |
Dweck D, Pinto JR, Reynaldo DP, Parvatiyar M, Jones MA, Liang J, Sorenson MM, Potter JD. Strong Crossbridges are Required to Recapitulate the Ca2+ Affinity Changes Produced by HCM-cTnC Mutants in Skinned Fibers Biophysical Journal. 98: 149a. DOI: 10.1016/J.Bpj.2009.12.802 |
0.738 |
|
| 2008 |
Dweck D, Hus N, Potter JD. Challenging current paradigms related to cardiomyopathies. Are changes in the Ca2+ sensitivity of myofilaments containing cardiac troponin C mutations (G159D and L29Q) good predictors of the phenotypic outcomes? The Journal of Biological Chemistry. 283: 33119-28. PMID 18820258 DOI: 10.1074/Jbc.M804070200 |
0.754 |
|
| 2005 |
Dweck D, Reyes-Alfonso A, Potter JD. Expanding the range of free calcium regulation in biological solutions. Analytical Biochemistry. 347: 303-15. PMID 16289079 DOI: 10.1016/J.Ab.2005.09.025 |
0.557 |
|
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