Rebecca L. Haines

Affiliations: 
MRC LMCB University College London, London, United Kingdom 
Area:
disease
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"Rebecca Haines"

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Sara E. Mole grad student 2005-2009 UCL
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Publications

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Haines RL, Lane EB. (2012) Keratins and disease at a glance. Journal of Cell Science. 125: 3923-8
Chen H, Common JE, Haines RL, et al. (2011) Wide spectrum of filaggrin-null mutations in atopic dermatitis highlights differences between Singaporean Chinese and European populations. The British Journal of Dermatology. 165: 106-14
Common JE, Brown SJ, Haines RL, et al. (2011) Filaggrin null mutations are not a protective factor for acne vulgaris. The Journal of Investigative Dermatology. 131: 1378-80
Pears MR, Codlin S, Haines RL, et al. (2010) Deletion of btn1, an orthologue of CLN3, increases glycolysis and perturbs amino acid metabolism in the fission yeast model of Batten disease. Molecular Biosystems. 6: 1093-102
Haines RL, Codlin S, Mole SE. (2009) The fission yeast model for the lysosomal storage disorder Batten disease predicts disease severity caused by mutations in CLN3. Disease Models & Mechanisms. 2: 84-92
Codlin S, Haines RL, Burden JJ, et al. (2008) Btn1 affects cytokinesis and cell-wall deposition by independent mechanisms, one of which is linked to dysregulation of vacuole pH. Journal of Cell Science. 121: 2860-70
Codlin S, Haines RL, Mole SE. (2008) btn1 affects endocytosis, polarization of sterol-rich membrane domains and polarized growth in Schizosaccharomyces pombe. Traffic (Copenhagen, Denmark). 9: 936-50
Kitzmüller C, Haines RL, Codlin S, et al. (2008) A function retained by the common mutant CLN3 protein is responsible for the late onset of juvenile neuronal ceroid lipofuscinosis. Human Molecular Genetics. 17: 303-12
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