Rebecca L. Haines - Publications
Affiliations: | MRC LMCB | University College London, London, United Kingdom |
Area:
diseaseYear | Citation | Score | |||
---|---|---|---|---|---|
2012 | Haines RL, Lane EB. Keratins and disease at a glance. Journal of Cell Science. 125: 3923-8. PMID 23104737 DOI: 10.1242/Jcs.099655 | 0.34 | |||
2010 | Pears MR, Codlin S, Haines RL, White IJ, Mortishire-Smith RJ, Mole SE, Griffin JL. Deletion of btn1, an orthologue of CLN3, increases glycolysis and perturbs amino acid metabolism in the fission yeast model of Batten disease. Molecular Biosystems. 6: 1093-102. PMID 20485751 DOI: 10.1039/B915670D | 0.665 | |||
2009 | Haines RL, Codlin S, Mole SE. The fission yeast model for the lysosomal storage disorder Batten disease predicts disease severity caused by mutations in CLN3. Disease Models & Mechanisms. 2: 84-92. PMID 19132115 DOI: 10.1242/Dmm.000851 | 0.684 | |||
2008 | Codlin S, Haines RL, Burden JJ, Mole SE. Btn1 affects cytokinesis and cell-wall deposition by independent mechanisms, one of which is linked to dysregulation of vacuole pH. Journal of Cell Science. 121: 2860-70. PMID 18697832 DOI: 10.1242/Jcs.030122 | 0.67 | |||
2008 | Codlin S, Haines RL, Mole SE. btn1 affects endocytosis, polarization of sterol-rich membrane domains and polarized growth in Schizosaccharomyces pombe. Traffic (Copenhagen, Denmark). 9: 936-50. PMID 18346214 DOI: 10.1111/J.1600-0854.2008.00735.X | 0.657 | |||
2008 | Kitzmüller C, Haines RL, Codlin S, Cutler DF, Mole SE. A function retained by the common mutant CLN3 protein is responsible for the late onset of juvenile neuronal ceroid lipofuscinosis. Human Molecular Genetics. 17: 303-12. PMID 17947292 DOI: 10.1093/Hmg/Ddm306 | 0.687 | |||
Show low-probability matches. |